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心力衰竭的分子生物学 被引量:3

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摘要 心力衰竭的分子生物学侯应龙1臧益民2(第四军医大学1唐都医院心内科7100382生理学教研室)关键词心力衰竭受体心脏在病理因素持续作用下,在历经肥厚并最终走向衰竭的病程中,心肌与心肌间质细胞基因表达与调控将发生一系列显著而复杂的异常变化,并势必从细胞...
机构地区 第四军医大学
出处 《心功能杂志》 1998年第2期96-97,共2页
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  • 2Teerlink JR, Massie BM. Beta-adrenergic blocker mortality trials in congestive heart failure. Am J Cardiol, 1999. 84: 94R.
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  • 4Frishman WH.Acute myocardial infarction:Role of β1 adrenergic blockers[J].Philadelphia,Lippincott-Raven,1996,pp. 1205-1214.
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  • 7Preffer MA,Braunwald E.Ventricular remodeling after myocardial infarction:experimental observation and clinical implication[J].Circulation,1990,81:1161.
  • 8Mcdonald KM.Angiotension-converting enzyme inhibition and beta-adrenocepter blockade regress established ventricular remodeling a canine model of distcrete myocardial damage[J].J Am Coll Cardiol,1994,24:1762-1764.
  • 9Preffer MA,Braunwald E,Moye LA,et al.Effect of Captopril on mortality and morbidity of patients with left ventricular dysfunction after myocardial infarction[J].N Engl J Med,1992,327:669.
  • 10ISIS-4 Collaborative Group.ISIS-4:a randomized factorial trial assessing early oral Captopril,oral Mononitrate,and intravenous magnesium sulfate in 58050 patients with suspected acute myocardial infarction[J].Lancet,1995,345:669.

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