电针猫足三里穴对下食管括约肌压力调节功能的影响

Effect of Electroacupuncture at "Zusanli" (ST 36) on Lower Esophageal Sphincter in the cats

[目的]研究电针(EA)猫足三里穴对下食管括约肌压力(LESP)的影响,探讨足三里穴与食管动力调节之间的关系。[方法]将28只健康家猫行内镜检查,确定无镜下食管病(炎)。随机分为足三里穴组、非经非络穴组。应用加拿大E-7-1-1-1-2-5-5-5型8通道细微注水测压管和荷兰UPS-2020型食管测压系统测定猫LESP。[结果]EA猫足三里穴和非经非络穴期间和停止后,LESP较EA前均有显著性升高,足三里穴组EA期间LESP由(16.21±5.49)mmHg(1mmHg=0.133kPa)升高至(23.14±8.81)mmHg(P<0.01),EA停止后LESP升至(25.50±6.73)mmHg(P<0.01);非经非络穴组EA期间LESP由(15.64±3.61)mmHg升至(18.93±5.57)mm-Hg(P<0.01),EA停止后LESP升至(20.43±5.24)mmHg(P<0.01)。EA足三里穴对LESP的升高程度在EA期间和停止后均明显高于非经非络穴(P<0.05)。[结论]EA健康猫足三里穴可以升高LESP,对下食管括约肌具有良性调理功能,其机制尚未完全阐明。

[Objective]To observe the impact of Zusanli point（st 36） electro-acupuncture（EA） on the pressure of lower esophageal sphincter pressure （LESP） of cats and to discuss the relation-ship between Zusanli point（st 36） and esophageal motility regulation. [Methods]After endoscope examination,28 cats were confirmed as healthy ones without esophagitis. All experimental cats were randomly divided into two groups： the Zusanli point（st 36） group and the non-channels noncollaterals points group. The LESP were measured and recorded by applying E-7-1-1-1-2-5-5-5 type 8 Channel Fine Infusion Piezometric Tube （Canada） and UPS-2020 type Esophageal Pressure Measurement System （Netherlands）. [Results]The LESP of Zusanli point（st 36） group and nonchannels non-collaterals points group rose significantly under and after EA than that of before EA. In Zusanli point（st 36） group, the LESP increased from（16. 21±5. 49）mmHg to（23. 14±8. 81）mmHg （P〈0. 01） during EA and reached（25.5±6.73）mmHg （P〈0. 01） after EA. In non-channels non-collaterals points group, the LESP increased from（15. 64±3. 61）mmHg to （18. 93±5.57）mmHg （P〈0.01） during EA and reached（20. 43±5.24）mmHg （P〈0. 01） after EA. The degree of LESP increase in Zusanli ooint（st 36） group was apparently greater than that of the non-channels non-collaterals points group （P〈0. 05）. [Conclusion]EA on Zasanli point （st 36） of healthy cats can cause obvious LESP rising and lead to wholesome regulation on KES. However, the mechanism remains unclear and need further studies.