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白介素-18的研究进展 被引量:3

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出处 《河北医学》 CAS 2009年第7期880-882,共3页 Hebei Medicine
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  • 1Kabayashi K,Clin Immunol Immunopathol,1997年,85卷,112页
  • 2Dao T,Cell Immunol,1996年,173卷,230页
  • 3Antonia MJ, Sui H, Vassiliki P, et al. In vivo retinal gene expression in early diabetes. IOVS, 2001, 42: 3047-3057.
  • 4周红鹰 梅妍 卢友光.RFDD—PCR建立人类疾病差异表达谱方法研究[J].中华医学遗传学杂志,2005,22:293-296.
  • 5McInnes IB, Gracie JA, Leung BP, et al. Interleukin 18: a pleiotropic participant in chronic inflammation. Immunol Today,2000, 21:312-315.
  • 6Tahara H, Oikawa Y, Shimada A, et al. Systemic Administration of IL-18 promotes diabetes development in young nonobese diabetic mice. J Immunol, 2003, 171: 5865-5875.
  • 7Esposito K, Nappo F, Giugliano F,et al. Cytokine milieu tends toward inflammation in type 2 diabetes (Letter). Diabetes Care,2003, 26:1647.
  • 8Aso Y, Okumura KI, Takebayashi K, et al. Relationships of plasma interleukin-18 concentrations to hyperhomocysteinemia and carotid intima-media wall thickness in patients with type 2diabetes. Diabetes Care,2003, 26:2622-2627.
  • 9Mohr S. Potential new strategies to prevent the development of diabetic retinopathy. Expert Opin Investig Drugs, 2004,13: 189-98.
  • 10Renhai C,Jacob F, Masashi K,et al. Interleukin-18 acts as an angiogenesis and tumor suppressor. The FASEB Journal, 1999 ,13: 2195-2202.

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二级引证文献14

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