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缝隙连接蛋白研究进展 被引量:1

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出处 《脑与神经疾病杂志》 2009年第4期319-320,F0003,共3页 Journal of Brain and Nervous Diseases
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  • 1Goodenough DA, Goliger JA, Paul DL. Connexins, nexons, and intercellular Communication. Annu Rev Biochem, 1996, 65: 475- 502.
  • 2Wilson MR, CloseTW, Trosko JE. Cell population dynamics ( apoptosis, mitosis, and cell-cellcommunication) during disruption of homeostasis. Exp Cell Res,2000,254:257-268.
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同被引文献31

  • 1成永霞,钟国强.缝隙连接与心房颤动的折返机制[J].医学综述,2006,12(6):335-336. 被引量:3
  • 2侯月梅,张小琴,乔峻,林仁勇,木拉提,卢晓梅,陈艳.风湿性心脏病持续性心房颤动患者右心耳缝隙连接蛋白40和43mRNA水平的研究[J].中国心脏起搏与心电生理杂志,2006,20(3):274-274. 被引量:2
  • 3金奇,吴立群.心肌连接蛋白在心房颤动发生机制中的作用[J].诊断学理论与实践,2007,6(3):266-268. 被引量:1
  • 4Alexander B, Charles A. Novel pharmacological targets for the rhythm control management of atrial fibrillation. Pharmacol Ther, 2011,132:300-313.
  • 5Jana R, Vladimir K, Tamara B, et al. Alterations of the intercel- lular coupling protein, connexin-43, during ventricular fibrillation and sinus rhythm restoration demonstrated in male and female rat hearts: A pilot study. Exp Clin Cardiol,2011,16 :116-120.
  • 6Benjamin FR, Jiaxiang Qu, Frank MV,et al. Phosphatase resist- ant gap junctions inhibit pathologic remodeling and prevent ar- rhythmias. Circ Res, 2011,108 : 1459-1466.
  • 7Euy-Myoung Jeong, Man Liu, Megan S, et al. Metabolic stress, reactive oxygen species, and arrhythmia. J Mol Cell Cardiol, 2012,52:454-463.
  • 8Xianming Lin, Joanna G, Aaron G, et al. Connexin40 and Con- nexin43 determine gating properties of atrial gap junction chan- nels. J Mnl Cell Cardiol,2010,48: 238.
  • 9Indra L, Robert PR, Xianming Lin, et al. Deletion of the last five C-terminal amino acid residues of onnexin43 leads to lethal ventricular airhythmias in mice ithout affecting coupling via gap junction channels. Basic Res Cardiol,2013,108 : 348.
  • 10Wetzel U, Boldt A, Lauschke J, et al. Expression of connexins 40 and 43 in human left atrium in atrial fibrillation of different ae- tiologies. Cardiovasc Med Heart, 2005,91:166-170.

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