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NIDDM患者尿白蛋白、转铁蛋白、N-乙酰-β-D氨基葡萄糖苷酶检测的临床意义及其关系探讨 被引量:4

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出处 《中国糖尿病杂志》 CAS CSCD 1998年第4期238-239,共2页 Chinese Journal of Diabetes
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二级参考文献4

  • 1陈上云,实用内科杂志,1991年,11卷,241页
  • 2Chung C K,Clin Chem,1989年,35卷,1672页
  • 3王晓东,中华医学杂志,1988年,68卷,215页
  • 4王晓南,中华肾脏病杂志,1985年,2卷,69页

共引文献48

同被引文献50

  • 1傅秀兰,陈庆荣,江永娣,董德长,陈明伟,林爱华,王桂兰.尿微量蛋白测定对肾脏病的临床意义[J].上海医学,1993,16(8):462-463. 被引量:20
  • 2朱国文,李熙建.肾脏功能不同程度损害时尿中几种微量蛋白与尿酶变化分析[J].国际检验医学杂志,2007,28(6):489-491. 被引量:6
  • 3Akihide T,Tetsuo H,Osamu K. Cell death and degeneration of renal proximal tubular cell in rats with subchronic cadmium intoxication [J]. Toxicol Pathol, 1993,21:341-351.
  • 4Griffin J L, Walker I. A, Troke T, et al. The initial pathogenesis of cadmium induced renal toxicity[J]. FEBS Lett, 2000,478 : 147-150.
  • 5Sabolie I, Herak-Kramberger C M, Brown D. Subchronic cadmium treatment affects the abundance and arrangement of cytoskeletal proteins in rat renal proximal tubule cells[J]. Toxicology, 2001,165 : 205-216.
  • 6Jurczuk M,Moniuszko-Jakoniukn J ,Brzooska M M. Involvement of some low molecular thiols in the peroxidative mechanisms of lead and ethanol action on rat liver and kidney[J]. Toxicology, 2006,219 ( 1 ) : 11-21.
  • 7Garlon G, Leleu B, Marez T M, et al. Biomonitoring of the adverse effects induced by the chronic exposure to lead and cadmium on kidney function: Usefulness of alpha-glutathione S-transferase[J]. Sci Total Environ, 2007,377(213) :165-172.
  • 8Thijssen S,Cuypers A, Maringw J,et al. Low cadmium exposure triggers a biphasie oxidative stress response in mice kidneys[J]. Toxicology,2007,236(1) :29-41.
  • 9De Flora S, Izzotti A D, Agostini F, et al. Mechanism of N-acetyl-cysteine in the prevention of DNA damage and cancer with special reference to smoking-related endpoints[J]. Carcinogenesis, 2001,22 (7) : 999.
  • 10Brzoska M M,Kaminski M, Supernak-bobko D, et al. Changes in the structure and function of the kidney of rats chronically exposed to cadmium. Ⅰ. Biochemical and histopathological studies[J]. Arch Toxicol, 2003,77(6) :344-352.

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