摘要
肾素—血管紧张素系统(RAS)完全涉及心血管功能的自身稳定性,其生理作用的主要调节剂是血管紧张素Ⅱ(AngⅡ)。AngⅡ的前体—血管紧张素肽原被特异的蛋白酶肾素水解,导致10个氨基酸的AngⅠ生成,另一种蛋白酶血管紧张素转换酶(ACE)从AngⅠ上水...
出处
《哈尔滨医科大学学报》
CAS
1998年第5期384-386,共3页
Journal of Harbin Medical University
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