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卒中后神经病理学与神经可塑性机制的分析 被引量:2

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摘要 卒中的发生不仅会导致脑受损区域神经功能的缺损,而且还可以致使远隔部位的神经细胞死亡。故而针对卒中,寻找更多、更好的防治措施,并对卒中后神经病理生理学方面的改变及神经可塑性机制进行探索,是十分必要的。
出处 《中国脑血管病杂志》 CAS 2010年第1期46-48,共3页 Chinese Journal of Cerebrovascular Diseases
基金 广西自然科学基金资助项目(桂科自0728133) 广西医科大学博士后流动站基金资助项目(20702011046)
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  • 1Lo EH, Dalkara T, Moskowitz MA. Mechanisms, challenges and opportunities in stroke[ J ]. Nat Rev Neurosci ,2003,4 (5) :399-415.
  • 2Lee SR,Wang X, Tsuji K, et al. Extracellular proteolytic pathophysiology in the neurovascular unit after stroke[ J]. Neural Res,2004,26 ( 8 ) : 854-861.
  • 3Hawkins BT, Davis TP. Tile blood brain harrier/neurovascular unit in health and disease [ J ]. Pharmacol Rev, 2005,57(2) :173-185.
  • 4Rao VR, Finkbeiner S. NMDA and AMPA receptors: old channels, new tricks [ J ]. Trends Neurosci, 2007,30 ( 6 ) : 284 -291.
  • 5Kriz J. Infamrnation in ischemic brain injury: timing is important [ J ]. Crit Rev Neurobiol,2006,18 ( 1/2 ) : 145-157.
  • 6Nighoghossian N, Wiart M, Berthezene Y. Novel applications of magnetic resonance imaging to image tissue inflammation after stroke [ J ]. J Neuroimaging, 2008, 18(4) :349-352.
  • 7Farooqui AA, Horrocks LA, Farooqui T. Modulation of inflammation in brain : a matter of fat [ J ]. J Neorocbem, 2007,101 (3) :577-599.
  • 8Rosell A, Ortega-Aznar A, Alvarez-Sabin J,et al. Increased brain expression of matrix metalloproteinase-9 after ischemic and hemorrhagic human stroke [ J ]. Stroke, 2006,37 (6) :1399-1406.
  • 9Nagase H, Visse R, Murphy G. Structure and function of matrix metalloproteinases and TIMPs [J ]. Cardiovasc Res, 2006,69 ( 3 ) : 562-573.
  • 10Vukasovie I, Tesija-Kuna A, Topic E, et al. Matrix metalloproteinases and their inhibitors in different acute stroke subtypes[ J]. Clin Chem Lab Med, 2006, 44 (4): 428-434.

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