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单核-巨噬细胞在急性呼吸窘迫综合征发病机制中的作用 被引量:1

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出处 《中国实用医药》 2010年第2期246-248,共3页 China Practical Medicine
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  • 1Beatrice B, Reto s, Thomas P, et al. Alveolar macrophages regulate neutrophfl reemitant in endotoxin-induced lung injury. Respir Res, 2005,6:61.
  • 2Sone Y, Serikov VB, Staub NC Sr, et al. Intravascular macrophage depletion attenuates endotoxin lung injury in anesthetized sheep. Appl Physiol, 1999,87 (4) : 1354-1359.
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  • 4李磊,王正国,胡承香,张玉纯.创伤血清异常蛋白对巨噬细胞功能作用的研究[J].中国危重病急救医学,1998,10(8):492-494. 被引量:11
  • 5Molloy RG, Manoick JA, Rodrick M s. Br J surg,1993,80:289.
  • 6Cao L, Liu C, Cai B, et al. Nuclear factor-kappa B expression in alveolar macrophages of mechanically ventilated neonates with respiratory distress, syndrome. Biol Neonate, 2004,86 (2) : 116-123.
  • 7Vancurova I, Bellani P, Davidson D. Activation of nuclear factorkappaβ and its suppression by dexamethasone in polymorphonuclear leukocytes : newborn versus adult Pediatr Res, 2001, 49 ( 2 ) : 257 -262.
  • 8Goannoudis PV Current eoncepts of the inflammatory response after major leaumar an update, injury, 2003.34(6) :397-404.
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二级参考文献1

  • 1Jürgen Bernhagen,Thierry Calandra,Richard Bucala. Regulation of the immune response by macrophage migration inhibitory factor: biological and structural features[J] 1998,Journal of Molecular Medicine(3-4):151~161

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