摘要
阿尔茨海默病(Alzheimer’S disease,AD)是一种病因未明,以记忆、认知受损为主要表现的中枢神经系统进行性退化性疾病。AD发病机制的各种假说中β淀粉蛋白(β-amyloid protein,Aβ)学说和tau蛋白学说得到较多的认可,但均不能完整地解释AD的发病过程。近来研究表明突触功能改变与认知的关系较为密切。
出处
《解剖学杂志》
CAS
CSCD
北大核心
2010年第1期125-127,共3页
Chinese Journal of Anatomy
基金
国家自然科学基金(30660073)
江西省自然科学基金(0640028)
江西省教育厅科研基(赣教技字[2005]188号)
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