1Rajalingam R, Parham PL. Abi-Rached. Domain shuffling has been the main mechanism forming new hominoid KIR. J Immunol,2004,172(1):356-369
2Karumanchi SA, Bdolah Y. Hypoxia and sFlt-1 in preeclampsia: the "chicken-and-egg" question.Endocrinology,2004,145(11):4835-4837
3Maynard SE, Min JY, Merchan J, et al. Excess placental soluble fmslike tyrosinekinase (sFlt1)may contribute to endothelial dysfunction,hypertension, and proteinuria in preeclampsia. J Clin Invest,2003,111(5):649-658
4Page NM, Woods RJ, Gardiner SM, et al. Excessive placental secretion of neurokinin B during the third trimester causes pre-eclampsia.Nature,2000,405(6788):797-800
5Redman CW, Sargent IL. Pre-eclampsia, the placenta and the maternal systemic inflammatory response-a review.Placenta,2003,24 [Suppl A]:S21-27
6Huppertz B, Kingdom J, Caniggia I, et al. Hypoxia favours necrotic versus apoptotic shedding of placental syncytiotrophoblast into the maternal circulation. Placenta, 2003,24(2-3): 181-190
7Raijmakers MT, Dechend R, Poston L. Oxidative stress and preeclampsia:rationale for antioxidant clinical trials.Hypertension,2004,44(4):374-380
8Sattar N, Greer IA. Pregnancy complications and maternal cardiovascular risk: opportunities for intervention and screening? BMJ,2002,325(7356):157-160
9van Dijk M, Mulders J, Poutsma A, et al. Maternal segregation of the Dutch preeclampsia locus at 10q22 with a new member of the winged helix gene family. Nat Genet,2005,37(5):514-519
10Lain KY, Roberts JM. Contemporary concepts of the pathogenesis and management of preeclampsia JAMA,2002,287(24):3183-3186
