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核转录因子-κB与酒精性肝病的研究进展 被引量:6

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出处 《临床肝胆病杂志》 CAS 2010年第3期269-271,共3页 Journal of Clinical Hepatology
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参考文献19

  • 1赵彩彦.核因子-κB与酒精性肝病[J].国外医学(消化系疾病分册),2002,22(4):230-232. 被引量:14
  • 2Diehl AM. Liver disease in alcohol abusers: clinical perspective [ J ]. Alcohol, 2002 ; 27 ( 1 ) : 7.
  • 3庄辉.酒精性肝病的流行病学[J].中华肝脏病杂志,2003,11(11):689-689. 被引量:202
  • 4Cheng Q, Lee HH, Li Y, el al. Upregulation of Bcl - x and Bfl - 1 as a potential mechanism of ehemoresistance, which can be overcome by NF-kappa B inhibition[ J]. Oncogene, 2000, 19(42): 4936 - 4940.
  • 5Wang CY, Guttridge DC, Mayo MW, et al. NF - kappa B induces expression of Bcl -2 homologue AI/Bf- 1 to preferentially suppress chemotherapy induced apoptosis[ J]. Mol Cell Biol, 1999, 19(9): 5923 - 5929.
  • 6Tumang JR, Hsia CY, Tian W, et al. IL-6 rescues the hyporesponsi veness of c - Rel deficient B cells independent of Bcl - xl, Mcl-1, and Bcl-2[J]. Celllmmunol, 2002, 217(1-2):47-57.
  • 7崔香丹,金武丕,李香.大鼠酒精性肝病细胞凋亡中Caspase-3、NF-κB的表达[J].陕西医学杂志,2009,38(3):273-275. 被引量:4
  • 8Hock JB, Pastofino JG. Ethanol, oxidative stress, and cytokine - induced liver cell injury [ J ]. AIcohol, 2002, 27 ( 1 ) : 63 - 68.
  • 9Kono H, Rusyn I, Uesugi T, et al. Diphenyleneiodonium sulfate, an NADPH oxidase inhibitor, prevents early alcoholinduced liver injury in the rat [ J ]. Am J Physiol Gastrointest Liver Physiol, 2001 , 280(5): 1005 - 1012.
  • 10Tomae Z, Malta K. Oxidative stress and signal transduction pathways in alcoholic liver disease [ J ]. Clinical and experimental, 2006, 29(11): 110 -115.

二级参考文献37

  • 1林春龙,张珍祥.FAK反义寡核苷酸促进人肺动脉平滑肌细胞凋亡[J].中国病理生理杂志,2006,22(2):288-291. 被引量:13
  • 2Setwart S, Jones D, Day CP. Alcoholic liver disease: mechanisms and preventative strategies [J]. Trendsnew insights in to Mol Med, 2001,7(9 ): 408-413.
  • 3Cheng Q,Lee HH,Li Y, et al. Upregulation of Bcl-x and Bfl-1 as a potential mechanism of chemoresistanee, which can be overcome by NF-kappa B inhibition [J]. Oncogene. 2000,19 (42) : 4936-4940.
  • 4Wang CY, Guttridge DC, Mayo MW, et al. NF-kappaB induces expression of Bcl-2 homologue A1/Bf-1 to preferentially suppress chemotherapy-induced apoptosis[J]. Mol Cell Biol,1999, 19(9):5923-5929.
  • 5Tumang JR, Hsia CY, Tian W et al. IL-6 rescues the hyporesponsi veness of c-Rel deficient B cells independent of Bcl-xl, Mcl-1, and Bcl-2 [ J ]. Cell Immunol,2002,217(1-2):47-57.
  • 6Lieber CS. Alcoholic liver disease: New insights in pathogenesis lead to new treatments. J Hepatol, 2000, 32:113-128.
  • 7Stewart SF, Day CP. The management of alcoholic liver disease. J Hepatol, 2003, 38: S2-S13.
  • 8Diehl AM. Alcoholic and nonalcoholic steatohepatitis. See: Arias IM, Boyer JL, Chisari FV, et al, eds. The Liver: Biology and Pathobiology. Fourth Edition. Lippincott Williams & Wilkins,Philadelphia, 2001. 739-753.
  • 9Garbutt JC, West SL, Carey TS, et al. Pharmacological treatment of alcohol dependence: a review of the evidence. JAMA, 1999, 281:1318-1325.
  • 10Mathurin P, Mendenhall CL, Carithers RL, et al. Corticosteroids improve short-term survival in patients with severe alcoholic hepatitis (AH): individual data analysis of the last three randomized placebo controlled double blind trials of corticosteroids in severe AH. J Hepatol, 2002, 36: 480-487.

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