1Virginie B, Aurelie N, Pierre M, et al. The role of pro- anti- inflammatory responses in silica-induced lung fibrosis [J]. Respir Res, 2005, 6 (1): 112.
3Jan IH, Vivi V, Per ES, etal. IL lbeta differently involved in IL-8 and FGF-2 release in crystalline silica treated lung cell cocultures [J]. Part Fibre Toxicol, 2008, 5: 16.
4Tara S, Maria R, Jeffrey B, et al. Gene expression profiles reveal increased mClea3 (GobS) expression and mucin production in a murine model of asbestos-induced fibrogenesis [J]. Am J Pathol, 2005, 167 (5): 1243-1256.
5Pamela G, Caroline M, Isabelle G, et al. IL-1R1/MyD88 signaling and the inflammasome are essential in pulmonary inflammarion and fibrosis in mice [J]. J Clin Invest, 2007, 117 (12):3786-3799.
6Webb TJ, Sumpter TL, Thiele AT, et al. The phenotype and function of lung dendritic cells [J]. Crit Rev Immunol, 2005, 25 (6): 465-491.
7Mei Z, Donald F, Aled P. ERK, p38, and smad signaling pathways differentially regulate transforming growth factor-β1 auto- induction in proximal tubular epithelial cells [J]. Am J Pathol, 2006, 169 (4): 1282-1293.
8Haining Y, Maurizio B, Barbara K, et al. TNF-a inhibits asbestos-induced cytotoxicity via a NF-κB-dependent pathway, a possible mechanism for asbestos induced oncogenesis [J]. Proc Natl Acad SciUSA, 2006, 103 (27): 10397-10402.
9Kim SH, Oh J, Choli JY, et al. Identification of human thioredoxin as a novel IFN gamma-induced factor: mechanism of induction and its role in cytokine production [J]. BMC Immunol, 2008, 9 (1): 64.
10David B, Silvestre V, Mario FF, et al. Molecular analysis of a multistep lung cancer model induced by chronic inflammation reveals epigenetic regulation of p16 and activation of the DNA damage response pathway [J]. Neoplasia, 2007, 9 (10): 840- 852.
2Hnizdo E, Vallyathan V. Chronic obstructive pulmonary disease due to occupational exposure to silica dust: a review of epidemiological and pathological evidence (Review). Occup Environ Med ,2003,60:237-243.
4Kaminski N, Rosas IO. Gene expression profiling as a window into idiopathic pulmonary fibrosis pathogenesis: can we identify the right target genes ( Review )? Proc Am Thorac Soc, 2006, 3:339-344.
5Kuhn K, Baker SC, Chudin E, et al. A novel, high-performance random array platform for quantitative gene expression profiling. Genome Res ,2004,14:2347-2356.
6Stokes TH, Han X, Moffitt RA, et al. Extending microarray quality control and analysis algorithms to illumina chip platform. Conf Proc IEEE Eng Med Biol Soc,2007:4637-4640.
7Osborne JD, Zhu LJ, Lin SM, et al. Interpreting microarray resuhs with gene ontology and MeSH (Review). Methods Mol Biol,2007, 377:223-242.
8Lu X,de la Pena L, Barker C, et al. Radiation-induced changes in gene expression involve recruitment of existing messenger RNAs to and away from polysomes. Cancer Res,2006,66 : 1052-1061.
9Lewin A ,Grieve IC. Grouping Gene Ontology terms to improve the assessment of gene set enrichment in microarray data. BMC Bioinformatics ,2006,3 : 426.
10Martin LD, Krunkosky TM, Dye JA, et al. The role of reactive oxygen and nitrogen species in the response of airway epithelium to particulates (Review). Environ Health Perspect, 1997, 105: 1301-1307.
