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非典型失神癫痫AY-9944模型的机制研究进展 被引量:1

Research progress on the mechanism of the AY-9944 model of atypical absence seizures
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出处 《中华神经外科疾病研究杂志》 CAS 2010年第5期472-474,共3页 Chinese Journal of Neurosurgical Disease Research
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参考文献24

  • 1Bercovici E,Cortez MA,Wang X,et al.Serotonin depletion attenuates AY-9944-mediated atypical absence seizures[J].Epilepsia,2006,47(2):240-246.
  • 2Nolan M,Bergazar M,Chu B,et al.Clinical and neurophysiologic spectrum associated with atypical absence seizures in children with intractable epilepsy[J].J Child Neurol,2005,20(5):404-410.
  • 3Bierkamper GG,Cenedella RJ.Induction of chronic epileptiform activity in the rat by an inhibitor of cholesterol synthesis,U18666A[J].Brain Res,1978,150(2):343-351.
  • 4Smith KA,Bierkamper GG.Paradoxical role of GABA in a chronic model of petit mal (absence)-like epilepsy in the rat[J].Eur J Pharmacol,1990,176(1):45-55.
  • 5Cortez MA,Cunnane SC,Snead OC 3rd.Brain sterols in the AY-9944 rat model of atypical absence seizures[J].Epilepsia,2002,43(1):3-8.
  • 6Cortez MA,McKerlie C,Snead OC 3rd.A model of atypical absence seizures:EEG,pharmacology,and developmental characterization[J].Neurology,2001,56(3):341-349.
  • 7Serbanescu I,Ryan MA,Shukla R,et al.Lovastatin exacerbates atypical absence seizures with only minimal effects on brain sterols[J].J Lipid Res,2004,45(11):2038-2043.
  • 8Persad V,Cortez MA,Snead OC 3rd.A chronic model of atypical absence seizures:studies of developmental and gender sensitivity[J].Epilepsy Res,2002,48(1-2):111-119.
  • 9Sooksawate T,Simmonds MA.Effects of membrane cholesterol on the sensitivity of the GABA(A) receptor to GABA in acutely dissociated rat hippocampal neurones[J].Neuropharmacology,2001,40(2):178-184.
  • 10Li H,Yao Z,Degenhardt B,et al.Cholesterol binding at the cholesterol recognition/interaction amino acid consensus (CRAC) of the peripheral-type benzodiazepine receptor and inhibition of steroidogenesis by an HIV TAT-CRAC peptide[J].Proc Natl Acad Sci USA,2001,98(3):1267-1272.

同被引文献32

  • 1JALLON P, LATOUR P. Epidemiology of idiopathic generalized epilepsies [ J ]. Epilepsia, 2005, 46 ( Suppl 9) : 10-14.
  • 2OLSSON I. Epidemiology of absence epilepsy. I. Concept and incidence [J]. Acta Paediatr Scand, 1988, 77(6): 860-866.
  • 3PANAYIOTOPOULOS C P. Idiopathic generalized epilepsies. In: PANAYIOTOPOULOS C P, ed. The Epilepsies : Seizures, Syndromes and Management [M]. Oxford, UK : Bladon Medical Publishing, 2005.
  • 4VRIELYNCK P. Current and emerging treatments for absence seizures in young patients [ J]. Neuropsychiatr Dis Treat, 2013, 9 : 963-975.
  • 5ALBINI M, MORANO A, FANELLA L, et al . Effectiveness of rufinamide in the treatment of idiopathic generalized epilepsy with atypical evolution : case report and review of the literature [ J ]. Clin EEG Neurosci, 2014, 23. pii: 1550059414559940.
  • 6PANAYIOTOPOULOS C P. Childhood absence epilepsy [ EP/ OL]. www. epilepsy, org/etf/childhood_absencehtml. 2000.
  • 7HIRSCH E, PANAYIOTOPOULOS C P. Childhood absence epilepsy and related syndromes. In: ROGER J, BUREAU M, DRAVET C, et al. Epileptic Syndromes in Infancy, Childhood and Adolescence [ M ]. 4th ed. Montrouge, France: John Libbey Eurotext Ltd, 2005.
  • 8BLUMENFELD H. CeUular and network mechanisms of spike- wave seizures [J]. Epilepsia, 2005, 46(Suppl 9): 21-33.
  • 9GAMBARDELLA A, LABATE A. The role of calcium channel mutations in human epilepsy [J]. Prog Brain Res, 2014, 213: 87 -96.
  • 10KLITTEN L L, MφLLER R S, NIKANOROVA M, et al. A balanced translocation disrupts SYNGAP1 in a patient with intellectual disability, speech impairment, and epilepsy with myoclonic absences (EMA) [ J ]. Epilepsia, 2011, 52 ( 12 ) : 190-193.

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