1Taylor W,Cladman D,Helliwell P,et al.Classification criteria forpsoriatic arthritis:development of new criteria from a large interna-tional study[J].Arthritis Rheum,2006,54(8):2665-2673.
3Dangoria NS, DeLay ML, Kingsbury DJ, et al. HLA-B27 misfolding is associated with aberrant intermolecular disulfide bond formation (dimerization) in the endoplasmic reticulum. J Biol Chem,2002, 277: 23459-23468.
4Marsh SGE, Albert ED, Bodmer WF, et al. Nomenclature for factors of the HLA system, 2002. Hum Immunol, 2002, 63: 1213-1268.
5Bird LA, Peh CA, Kollnberger S, et al. Lymphoblastoid cells express HLA-B27 homodimers both intracellularly and at the cell surface following endosomal recycling. Eur J Immunol, 2003, 33:748-759.
6Malik P, Klimovitsky P, Deng LW, et al. Uniquely conformed peptide-containing beta 2-microglobulin-free heavy chains of HLA-B2705 on the cell surface. J Immunol, 2002, 169: 4379-4387.
7Boyson JE, Erskine R, Whitman MC, et al. Disulfide bond-mediated dimerization of HLA-G on the cell surface. Proc Natl Acad Sci USA, 2002, 99: 16180-16185.
8Ringrose JH. HLA-B27 associated spondyloarthropathy, an autoimmune disease based on cross reactivity between bacteria and HLA-B27. Ann Rheum Dis, 1999, 58: 598-610.
9May E, Dorris ML, Satumtira N, et al. CD8+T cells are not essential to the pathogenesis of arthritis or colitis in HLA-B27 transgenic rats. J Immunol, 2003, 170: 1099-1105.
10Trowsdale J. Genetic and functional relationships between MHC and NK receptor genes. Immunity, 2001, 15: 363-374.