肺炎支原体肺炎发病机制研究进展被引量：138

Research progression of pathogenesis of Mycoplasma pneumoniae pneumonia

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摘要肺炎支原体(MP)是一种介于细菌与病毒之间的微小非活性生长的微生物。MP没有细胞壁,有膜及胞浆抗原,主要的膜抗原是糖脂质。MP是目前支气管炎、肺炎的重要病原体之一,并且可导致血液、神经、消化、泌尿、循环等多系统及皮肤的病变。肺炎支原体肺炎(MPP)是小儿肺炎的常见类型之一。近年来,随着病原学变迁,MP已成为小儿呼吸道感染的重要病原体之一,并已成为学龄期儿童社区获得性肺炎(CAP)的常见病原体。迄今MPP的发病机制仍不十分清楚,目前国内外学者主要倾向于MP的黏附、MP的侵入、MP直接造成细胞损伤及免疫炎症反应。 Mycoplasma pneumoniae（MP）is a genus of tiny incompetence-growing microorganism between bacteria and virus.Without cell wall,MP has membrane and cytoplasmic antigens.Its main membrane antigen is glycolipid.MP is a humanpathogenic bacteria and is one of the important pathogens of bronchitis and pneumonia,which can lead to multi-system lesions of blood,nerve,digestion,urinary,circulation and lesion of skin.Mycoplasma pneumoniae pneumonia（MPP）is a common type of pneumonia in children.With the changes of the etiology,MP has recently become an important pathogen of respiratory infection in children as well as the common pathogen of community-acquired pneumonia of the school-age children.To date,the pathogenesis of MPP has not clear yet,the main pathogenesis tends to be adhesion of MP,penetration of MP,direct injury to cells and immune inflammation reaction.

作者刘洋李敏徐佩茹

机构地区新疆医科大学第一临床医学院儿科

出处《临床儿科杂志》 CAS CSCD 北大核心 2011年第2期196-198,共3页 Journal of Clinical Pediatrics

关键词肺炎肺炎支原体发病机制 pneumonia Mycoplasma pneumoniae pathogenesis

分类号 R725.6 [医药卫生—儿科]

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