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缬沙坦治疗老年性心力衰竭的分子生物学机制 被引量:2

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摘要 老年性心力衰竭是常见的老年性疾病,发病率及死亡率均很高。其发生、发展的机制是心脏重塑,而交感神经系统和肾素-血管紧张素系统(RAS)的过度激活则导致心力衰竭进行性恶化,血管紧张素Ⅱ(AngⅡ)是RAS最重要的血管活性物质,通过与受体结合发挥作用。为此,就血管紧张素Ⅱ受体拮抗剂(ARB)治疗老年性心力衰竭的作用机制和应用进展简述如下。
出处 《检验医学与临床》 CAS 2011年第6期736-737,共2页 Laboratory Medicine and Clinic
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参考文献11

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共引文献80

同被引文献18

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