1Cutler RG, Rodriguz H. Critical review of oxidative stress andAging[M]. London,World Sci Pub Co Pte Ltd, 2003: 3-23.
2Harman D. Aging: A theory based on free radical and radiation chemistry[J]. J Gerontol, 1956 ; 11:289-300.
3Yu BP. Aging and oxidative stress :Modulation by dietary restriction [J]. Free Radic Biol Med, 1996 ; 21 : 651-668.
4Ho YS,Magnenat JL,Bronson RT,et al. Mice deficient in cellular glutathione peroxidase develop normally and show no increased sensitivity to hyperoxia [J]. J Biol Chem, 1997: 272. 16644-16651.
5Barja G. Rate of generation of oxidative stress-related damage and animal longevity[J]. Free Radic Biol Med, 2002 ; 33 : 1167-1172.
6Litchfield JE,Thorpe SR,Baynes JW. Oxygen is not required for the browning and crosslinking of protein by pentoses: Relevance to Maillard reactions in vivo[J]. Int J Biocem Cell Biol, 1999; 31:1297-1305.
7Yin D, Bruk UT. Carbonyl toxification hypothesis of biological aging. In Maeieira-Coelho A. (ed.) Molecular basis of aging[M].New York :CRC Press Ine, 1995 : 421-436.
8Masoro EJ. (ed.) Handbook of physiology, Section 11: Aging[M]. Oxford :Oxford University Press, 1995 : 1-234.
9Cerami A. Hypothesis: Glucose as a mediator of aging [J]. J Am Geriatr Soc, 1985;33 : 626-634.
10Kristal BS, Yu BP. An emerging hypothesis : Synergistic induction of aging by free radicals and Maillard reactions [J]. J Cerontol,1992;47:B107-B114.
