1Rozzman K, Klaassen C. Absorption, distribution and excretion of toxicants. In: Klaassen C, ed. Caserett and Doull's Toxicology: Basic Science of Poisons. New York: McGraw Hill, 2001: 107- 132.
2di Bethizy JD, Hayes J. Metabolism: a determinant of toxicity. In: Hayes AW, ed. Principles and Methods of Toxicology. Philadelphia, PA:Taylor and Francis, 2001 :77-136.
3Mehendale HM. Tissue repair: an important determinant of final outcome of toxicant induced injury. Toxicol Pathol, 2005, 33:41- 51.
5Shankar K, Vaidya VS, Corton JC, et al. Activation of PPAR alpha in streptozotocin-induced diabetes is essential for resistance against acetaminophen toxicity. FASEB J, 2003, 17: 1748-1750.
6Shankar K, Vaidya VS, Apte UM, et al. Type 1 diabetic mice are protected from acetaminophen hepatotoxieity. Toxicol Sei, 2003, 73: 220-234.
7Shankar K, Vaidya VS, Wang T, et al. Streptozotocin-induced diabetic mice are resistant to lethal effects of thioacetamide hepatotoxicity. Toxicol Appl Pharmacol, 2003, 188 : 122-134.
8Anand SS, Murthy SN, Vaidya VS, et al. Tissue repair plays pivotal role in final outcome of liver injury following chloroform and allyl alcohol binary mixture. Food Chem Toxicol, 2003, 41: 1123-1132.
9Anand SS, Soni MG, Vaidya VS, et al. Extent and timeliness of tissue repair determines the dose related hepatotoxicity of chloroform. Int J Toxicol, 2003, 22:25-33.
10Apte UM, Limaye PB, Desaiah D, et al. Mechanisms of increased liver tissue repair and survival in diet restricted rats treated with equitoxic doses of thioacetamide. Toxicol Sci, 2003, 72 : 272-282.
