期刊文献+

UF-100i全自动尿有形成份分析仪在脑脊液细胞分析中的应用 被引量:2

The Application of the UF-100i Fully Automated Urine Cell Analyzer in the Cells in the Cerebrospinal Fluid
下载PDF
导出
摘要 目的 探讨UF-100i全自动尿有形成份分析仪在脑脊液细胞分析中的应用.方法 无菌采集脑脊液后,用UF-100i全自动尿有形成份分析仪对标本进行分析及传统手工方法分别计数脑脊液细胞,对计数结果进行统计学分析.结果 UF-100全自动尿有形成份分析仪检测红细胞、白细胞与显微镜目测法计数经配对t检验,无显著差异.结论 全自动尿有形成份分析仪计数脑脊液中的细胞快速、结果准确且重复性好,适合临床常规检测脑脊液标本. Objective To discuss the application of the UF-100i fully automated urine cell analyzer in the cells in the cerebrospinal fluid. Methods We made a collection of the asepsis cerebrospinal fluid, and counted the cells number with the UF-100i fully automated urine cell analyzer and the traditional method. Results The test match t (α=0. 05),there is no marked difference in meaning and sexual.UF-100i fully automated urine cell analyzer can be considered in the test of the red cells,white cells and get a law of the counting have no significant differences. Conclusion It is very quick,accurate and good repeatability of the UF-100i fully automated urine cell analyzer counting the cells in the cerebrospinal fluid.It is fit for regular inspection and clinical specimens of cerebrospinal fluid.
出处 《中国血液流变学杂志》 CAS 2011年第1期158-159,163,共3页 Chinese Journal of Hemorheology
关键词 脑脊液 UF-100i全自动尿有形成份分析仪 cerebrospinal fluid UF-100i fully automated urine cell analyzer
  • 相关文献

参考文献5

二级参考文献57

  • 1[1]Ross R. Atherosclercsis: an inflammatory disease.disease. N Engl J Med, 1999,340:115-126
  • 2[2]PasceriV, Willerson JT, Yeh ETH. Direct proinflammatory effect of C-reactive protein on human endothelial cells. Circulation, 2000, 102:2 165-168
  • 3[3]Pasceri V, Chang J, Willerson JT, etal. Modulation of C-reactiveprotein-mediated monocyte chemoattractant protein-1 induction in human endothelial cells by anti-atherosclemsis drugs. Circulation, 2001. 103:2 531-534
  • 4[4]Zwaka TP, Hombach V, Torzewski J. C-reactive protein-mediated low density lipoprotein uptake by macrophages implications for atherosclerosis. Circulation,2001, 103: 1194-197
  • 5[5]Bhakdi S, Torzewski M, Klouche M, et al. Complement and athemgenesis:binding of CRP to degraded, nonoxidized LDL enhances complement activation.Arterisocler Thromb Vasc Biol, 1999, 19:2 348-354
  • 6[6]Griselli BM, Herbert J, Hutchirnson WL, et al. C-eractive protein and complement are important mediators of tissue damage in acute myocardial infarction. J Exp Med, 1999, 190: 1733-739
  • 7[7]Zhang YX, Cliff WJ, Schoefl GI, et al. Coronary C-reactive protein distribution: its relation to development of atherosclerosis. Atherosclerosis, 1999, 145:375-379
  • 8[8]HeinrichJ, Suchute H, Schonfeld R, et al. Association of variables of coagulation, fibrinolysis and acute-phase with atherosclerosis in coronary and peripheral arteries and those arteries supplying the brain. Thrornb Haemostas, 1995, 73:374-378
  • 9[9]Ridker PM, Stampfer MJ, Rifai N. Novel risk factors for systemic atherosclerosis: a comparison of C-reactive protein, fibrinogen, homocysteine, lipoprotein (a), and standard cholesterol screening as predictors of preipheral arterial diseaes. JAMA, 2001, 285:24 814-815
  • 10[10]Tataru MC, Heinrich J, Junker R, et al. C-reactive protein and the severity of atherosclerosic in myocardial infaction patients with stable angina pertoris. Eur heart J, 2000, 21:1000

共引文献59

同被引文献15

引证文献2

二级引证文献6

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部