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先天性QT延长综合征的遗传学基础及临床意义 被引量:3

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作者 耿茜
出处 《中国心脏起搏与心电生理杂志》 1999年第4期251-253,共3页 Chinese Journal of Cardiac Pacing and Electrophysiology
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参考文献6

  • 1Wang D W,J Clin Invest,1997年,99卷,7期,1714页
  • 2Lee M P,Nature Genetics,1997年,15卷,2期,181页
  • 3Compton S J,Circulation,1996年,94卷,5期,1018页
  • 4Wang Q,Genomics,1996年,34卷,1期,9页
  • 5Sanguinetti M C,Cell,1995年,81卷,2期,299页
  • 6Ko Y L,Human Genet,1994年,94卷,4期,364页

同被引文献16

  • 1方圻,王思让.心律失常的临床对策[J].中华心血管病杂志,1993,21(1):5-14. 被引量:39
  • 2张丽琴,曾碧英.长Q-T综合征的Q-T离散度[J].临床心电学杂志,1997,6(1):25-25. 被引量:2
  • 3[3]Wang Q,Curran ME,Splawski L,et al. Positional cloning of novel potassium channel gene: KVLQT1 mutations cause cardiac arrhythmis. Nat Genet, 1996,12:17
  • 4[4]Benson DW, Macrae CA,Vesely MR,et al. Missense mutation in the pore region of HERG causes familial long QT syndrome. Circulation, 1996,93:1791
  • 5[5]Wang Q,Shen JX,Li ZZ,et al. Cardiac sodium channel mutation in patients with long QT syndrome,an inherited cardiac arrhythmia. Hum Mol Genet, 1995,4:1063
  • 6[7]Tan HL, Hou CJY, Lauer MR, et al. Electrophysiologic mechanisms of the long QT interval syndrome and trosede de pointes. Ann Intern Med, 1995,122: 701
  • 7[8]Burashnikov A, Antzelevitch C. Bete-adrenergic stimulation produces transient action potential prolongation in canine ventricular M cells but not in purkinje, epicardial,or endocardial cell when contribution of Ikr is reduced(abstract).PACE, 1996,19:293
  • 8[9]Schwartz PJ,Moss AJ,Vincent GM,et al. Diagnostic criteris for the long QT syndrome on update. Circulation, 1993,88:782
  • 9[10]Barry JM,Chair JHM,Christine ES,et al. Impact of laboratory molecular diagnosis on contemporary diagnostic criteria for genetically transmitted cardiovascular disease: hypertrophic cardiomy-opathy, long-QT syndrome, and marfan syndrome. Circulation, 1998,98:1460
  • 10[11]Compton SJ, Lux PL, Ramsey MR, et al. Genetically defined therapy of inherited long QT syndrome:Correction of abnormal pepolarization by potassium. Circulation, 1996,94:1018

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