3[3]Wang Q,Curran ME,Splawski L,et al. Positional cloning of novel potassium channel gene: KVLQT1 mutations cause cardiac arrhythmis. Nat Genet, 1996,12:17
4[4]Benson DW, Macrae CA,Vesely MR,et al. Missense mutation in the pore region of HERG causes familial long QT syndrome. Circulation, 1996,93:1791
5[5]Wang Q,Shen JX,Li ZZ,et al. Cardiac sodium channel mutation in patients with long QT syndrome,an inherited cardiac arrhythmia. Hum Mol Genet, 1995,4:1063
6[7]Tan HL, Hou CJY, Lauer MR, et al. Electrophysiologic mechanisms of the long QT interval syndrome and trosede de pointes. Ann Intern Med, 1995,122: 701
7[8]Burashnikov A, Antzelevitch C. Bete-adrenergic stimulation produces transient action potential prolongation in canine ventricular M cells but not in purkinje, epicardial,or endocardial cell when contribution of Ikr is reduced(abstract).PACE, 1996,19:293
8[9]Schwartz PJ,Moss AJ,Vincent GM,et al. Diagnostic criteris for the long QT syndrome on update. Circulation, 1993,88:782
9[10]Barry JM,Chair JHM,Christine ES,et al. Impact of laboratory molecular diagnosis on contemporary diagnostic criteria for genetically transmitted cardiovascular disease: hypertrophic cardiomy-opathy, long-QT syndrome, and marfan syndrome. Circulation, 1998,98:1460
10[11]Compton SJ, Lux PL, Ramsey MR, et al. Genetically defined therapy of inherited long QT syndrome:Correction of abnormal pepolarization by potassium. Circulation, 1996,94:1018