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辛伐他汀对非酒精性脂肪肝患者血清视黄醇结合蛋白4水平的影响

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摘要 非酒精性脂肪肝（non-alcoholic fatty liver disease,NAFLD）是一种无过量饮酒史致肝实质细胞脂肪变性和脂肪贮积、炎症和纤维化为特征的疾病,与肥胖、高脂血症的发生密切相关[1]。
作者 张青波
机构地区 山西省公安消防总队医院
出处 《山西医药杂志（上半月）》 CAS 2011年第7期691-693,共3页 Shanxi Medical Journal
关键词 非酒精性脂肪肝 视黄醇结合蛋白4 脂肪肝患者 辛伐他汀 肝实质细胞脂肪变性 血清 liver 脂肪贮积
分类号 R575.5 [医药卫生—消化系统]
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参考文献10

  • 1Yang ZX, Shen W, Sun H. Effects of nuclear receptor FXR on the regulation of liver lipid metabolism in patients with non-alcoholic fatty liver disease. Hepatol Int, 2010,4 (4) : 741 -748.
  • 2Koch A, Weiskirchen R, Sanson E, et al. Circulating retinol binding protein 4 in critically ill patients before specific treat- ment: prognostic impact and correlation with organ func- tion, metabolism and inflammation. Crit Care, 2010,14 ( 5 ) R179.
  • 3Ahmed MH, Byrne CD. Current treatment of non-alcoholic fatty liver disease. Diabetes Obes Metab, 2009,11 ( 3 ) : 188- 195.
  • 4Dowman JK, Tomlinson JW, Newsome PN. Pathogenesis of non alcoholic fatty liver disease. QJM, 2010,103(2):71-83.
  • 5Su YX, Hong J, Yan Q, et al. Increased serum retinol-bind ing protein-4 levels in pregnant women with and without ges tational diabetes mellitus. Diabetes Metab, 2010, 36 (6 Pt 1) :470-475.
  • 6Bobbert P, Weithfiuser A, Andres J, et al. Increased plasma retinol binding protein 4 levels in patients with inflammatory cardiomyopathy. Eur J Heart Fail,2009,11(12) :1163-1168.
  • 7Seo JA, Kim NH, Park SY, et al. Serum retinol-binding protein 4 levels are elevated in non-alcoholic fatty liver dis ease. Clin Endocrinol (Oxf) ,2008,68(4) :555-560.
  • 8Schina M, Koskinas J, Tiniakos D, et al. Circulating andliver tissue levels of retinol-binding protein-4 in non-alcoholic fatty liver disease. Hepatol Res,2009,39(10) :972-978.
  • 9Takebayashi K, Suetsugu M, Matsumoto S, et al. Effects of rosuvastatin and colestimide on metabolic parameters and urinary monocyte chemoattractant protein-1 in type 2 diabet- ic patients with hyperlipidemia. South Med J,2009,102(4): 361-368.
  • 10Pftitzner A, Schondorf T, Hanefeld M, et al. Changes in in-sulin resistance and cardiovascular risk induced by PPAR- gamma activation have no impact on RBP4 plasma concentra- tions in nondiabetic patients. Horm Metab Res, 2009, 41 (3) :202-206.
山西医药杂志（上半月）
2011年 第7期

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