1Hsieh CC, Yen MH, Liu HW, et al. Lysophosphatidylcholine induces apoptofic and non-apoptotic death in vascular smooth muscle cells: in comparison with oxidized LDL [ J ]. Atherosclerosis, 2000,151 ( 2 ) :481 -491.
2Grey ST, Arvelo MB, Hasenkamp W, et al. A20 inhibits cytokine-induced apoptosis and nuclear factor kappa B-dependent gene activation in islets[J]. J Exp Med, 1999,190(8) :1135 -1146.
3Lee EG, Boone DL, Chai S, et al. Failure to regulate TNF-induced NF- kappaB and cell death responses in A20-deficient mice [ J ]. Science, 2000,289 (5488) :2350 - 2354.
4Heyninck K, Kreike MM, Beyaert R. Structure-function analysis of the A20-binding inhibitor of NF-kappa B activation, ABIN-1 [ J ]. FEBS Lett, 2003,536(1-3) :135 - 140.
5Coomaert B, Carpentier I, Beyaert R. A20: central gatekeeper in inflammation and immunity [ J ]. J Biol Chem, 2009,284 ( 13 ) : 8217 - 8221.
6Subramanian S V,Kelm RJ,Polikandriotis J A,et al.Reprogramming of vascular smooth muscle α-actin gene expression as an early indicator of dysfunctional remodeling following heart transplant[J].Cardiovasc Res,Jun 2002; 54(3):539-548.
7Stegemann J P,Hong H,Nerem R M.Mechanical,biochemical,and extracellular matrix effects on vascular smooth muscle cell phenotype[J].Appl Physiol,2005,98(6):2321-2327.
8Takahashi M,Hayashi K,Yoshida K,et al.Epiregulin as a major autocrine/paracrine factor released from ERK and p38MAPK-activated vascular smooth muscle cells[J].Circulation,2003,108(20):2524-2529.
9Yanagisawa M,Inoue A,Ishikawa T,et al.Primary structure,synthesis,and biological activity of rat endothelin,an endothelium-derived vasoconstrictor peptide[J].Proc Natl Acad Sci U S A,1988,85(18):6964-6967.
10Wang C Y,Zhang Y L,Yang Q,et al.A novel cultured tissue model of rat aorta:VSMC proliferation mechanism in relationship to atherosclerosis[J].Exp Mol Pathol,2007,83(3):453-445.
2Jt Subramanian P, Karshovska E, Reinhard P, et al. Iysophospha- tidic acid receptors LPA1 and LPA3 promote CXCLl2-mediated smooth muscle progenitor cell recruitment in neointima formation [J]. Circ Res,2010,107(1):96-105.
3Zernecke A,Schober A,Bot I,et al. SDF-lalpha/CXCR4 axis is instrumental in neointimal hyperplasia and recruitment of smooth muscle progenitor cells[J]. Circ Res,2005,96(7):784-791.
4Yokoi H,Yamada H,Tsubakimoto Y,et al. Bone marrow ATI augments neointima formation by promoting mobilization of smooth muscle progenitors via platelet-derived SDF-I {alpha}[J]. Arte- rioscler Thromb Vasc Biol, 2010,30(1) : 60-67.
5Campagnolo P,Wong MM,Xu Q. Progenitor Cells in Arterioscle- rosis : good or bad Guys [ J ]. Antioxid Redox Signal, 2011,15 (4) : 1013-1027.
6Caplice NM,Bunch TJ,Stalboerger PG,et al. Smooth muscle cells in human coronary atherosclerosis can originate from cells admin- istered at marrow transplantation [J]. Proc Natl Acad Sci U S A, 2003, 100(8):4754-4759.
7Sata M,Saiura A,Kunisato A,et al. Hema topoietic stem cells differentiate into vascular cells that participate in the pathogenesis of atherosclerosis[J]. Nat Med,2002,8(4):403-409.
8Han CI, Campbell GR, Campbell JH. Circulating bone marrow cells can contribute to neointimal formation [J]. J Vasc Res,2001,38 (2):113-119.
9Hillebrands JL,Klatter FA,vanden Hurk BM,et al. Origin of neointimal endothelium and alpha-actin-positive smooth muscle cells in transplant arteriosclerosis [J]. J Clin Invest,2001,107 (11): 1411-1422.
10Shimizu K,Sugiyama S,Aikawa M,et al. Host bone-marrow cells are a source of donor intimal smooth-muscle-like cells in murine aortic transplant arteriopathy [J]. Nat Med,2001,7(6):738-741.
