期刊文献+

微创穿刺治疗高血压基底节脑出血水肿带变化的临床疗效观察 被引量:2

Clinical effective study of edema zone change of hypertensive basal ganglia hemorrhage treated by minimally invasive puncture
下载PDF
导出
摘要 目的:探讨高血压性基底节脑出血微创穿刺和尿激酶注入的临床效果。方法:应用YL-1型颅内血肿穿刺针脑内血肿穿刺并持续引流,术后用尿激酶注入血肿腔,通过颅脑CT检查,与保守治疗对比,观察发病后同一天水肿带变化和拔管前后水肿带的变化。结果:两组病人入院时水肿带差异无统计学意义(P>0.05),治疗组病人术后水肿带较术前明显减轻,入院第一、四、五、九天明显低于对照组,差异有统计学意义(P<0.01)。结论:微创穿刺治疗高血压基底节脑出血并尿激酶注入及持续引流,疗效明确,脑组织损伤小,并发症少,预后好。 Objective:To explore the clinical efficacy of minimally invasive puncture,injecting urokinase and drainage in the treatment of hypertensive basal ganglia hemorrhage. Methods:The YL-1 type intracranial hematoma puncture needle was used to perform intracranial hematoma puncture and sustained external drainage. After operation, urokinase was injected into hematoma cavity. By cerebral CT examination,the edema zone change at the same day after onset,before and after extubation were observed and compared with that in the conservative treatment.Results:The edema zone had no statistical difference between the two groups(P〉0.05).The edema zone after op- eration in the treatment group was significantly alleviated than that before operation. The edema zone on 1,4,5,9 d after admission in the treatment group was obviously lower than that in the control group with statistical significance (P〈0.01).Conclusion: Minimally invasive puncture combined with urokinase injection and continued external drainage is effective to treat hypertensive basal ganglia hemorrhage with less brain injury, less complications and better prognosis.
机构地区 日照市中医医院
出处 《现代医药卫生》 2011年第19期2890-2891,共2页 Journal of Modern Medicine & Health
关键词 高血压 基底节脑出血 微创穿刺 尿激酶 Hypertension Basal ganglia cerebral hemorrhage Minimally invasive puncture Urokinase Edema zone value Drainage Before and after extubation Hematoma elimination
  • 相关文献

参考文献4

二级参考文献24

  • 1陈恒年,王绍鹏,王振金,赵坤,李英杰.超早期脑血肿碎吸引流术治疗高血压性脑出血[J].中华神经外科杂志,1993,9(1):37-39. 被引量:141
  • 2贾保祥,孙仁泉,顾征,徐屹,孙永权.穿刺射流及液化技术治疗高血压脑出血的初步报告[J].中国神经精神疾病杂志,1996,22(4):233-235. 被引量:665
  • 3[1]NINDS Workshop Participants.Priorities for Clinical Research in Intracerebral Hemorrhage:Report From a National Institute of Neurologic Disorders and Stroke Workshop[J].Stroke,2005,36:23-41.
  • 4[2]Davis SM,Broderick J,Hennerici M,et al.Hematoma growth is a determinant of mortality and poor outcome after intracerebral hemorrhage[J].Neurology,2006,66:1175-1181.
  • 5[3]Brott T,Broderick J,Kothari R,et al.Early hemorrhage growth in patients with intracerebral hemorrhage[J].Stroke,1997,28:1-5.
  • 6[4]Mayer SA,Brun NC,Begtrup K,et al.Recombinant activated factor Ⅶ for acute intracerebral hemorrhage[J].N Engl J Med,2005,352:777-854.
  • 7[5]Willmot M,Leonardi-Bee J,Bath PMW.High blood pressure in acute stroke and subsequent outcome:a systematic review[J].Hypertension,2004,43:18-24.
  • 8[6]Broderick JP,Adams HP Jr,Barsan W,et al.Guidelines for the management of spontaneous intracerebral hemorrhage:a statement for healthcare professionals from a special writing group of the Stroke Council,American Heart Association[J].Stroke,1999,30:905-915.
  • 9[7]Juvela S,Heiskanen O,Poranen A,et al.The treatment of spontaneous intracerebral hemorrhage:a prospective randomized trial of surgical and conservative treatment[J].J Neurosurg,1989,70:755-758.
  • 10[8]Mendelow AD,Gregson BA,Fernandes HM,et al.Early versus initial conservative treatment in patients with spontaneous supratentorial intracerebral haematomas in the International Surgical Trial in Intracerebral Haemorrhage (STICH):a randomised trial[J].Lancet,2005,365:387-397.

共引文献227

同被引文献15

引证文献2

二级引证文献3

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部