期刊文献+

干细胞的预适应研究进展

原文传递
导出
摘要 通过移植外源干/祖细胞或促进内源性的干细胞更多参与组织的修复过程是缺血损伤后的心肌再生治疗。10余年前开始使用源于骨骼肌的单一潜能的心肌前体细胞和终末分化心肌细胞,
出处 《中华医学杂志》 CAS CSCD 北大核心 2011年第48期3452-3454,共3页 National Medical Journal of China
  • 相关文献

参考文献1

二级参考文献199

  • 1Zhao ZQ,Corvera JS,Halkos ME,et al.Inhibition of myocardial injury by ischemic postconditioning during reperfusion: comparison with ischemic preconditioningAmerican Journal of Physiology - Heart and Circulatory Physiology,2003.
  • 2Cadenas E,Davies KJ.Mitochondrial free radical generation, oxidative stress, and agingFree Radical Biology and Medicine,2000.
  • 3Green D R,Kroemer G.The pathophysiology of mitochondrial cell deathScience,2004.
  • 4Murphy MP.How mitochondria produce reactive oxygen speciesBiochemical Journal,2009.
  • 5Basso E,Petronilli V,Forte MA,Bernardi P.Phosphate is es-sential for inhibition of the mitochondrial permeability tran-sition pore by cyclosporin A and by cyclophilin D ablationJournal of Biological Chemistry,2008.
  • 6Roberto Bolli.Mechanism of myocardial "Stunning",1990.
  • 7Forbes RA;Charles SC;Murphy E.Diazoxide-induced cardioprotection requires signaling through a redox-sensitive mechanism,2001.
  • 8Hunter D R;Haworth R A;Southard J H.Relationship between configuration, function, and permeability in calcium-treated mitochondria,1976(16).
  • 9Michael V. Cohen MD,Xi-Ming Yang,James M. Downey.Acidosis, oxygen, and interference with mitochondrial permeability transition pore formation in the early minutes of reperfusion are critical to postconditioning’s success[J]. Basic Research in Cardiology . 2008 (5)
  • 10Brown GC,Cooper CE.Nanomolar concentrations of nitric oxide reversibly inhibit synaptosomal respiration by competing with oxygen at cytochrome oxidase. FEBS Letters . 1994

共引文献64

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部