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原发性醛固酮增多症106例患者回顾性分析 被引量:3

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摘要 原发性醛固酮增多症(primary aldosteronism,PA)指肾上腺腺瘤、肾上腺皮质增生等几种病理改变所造成的高血压伴有低钾血症、低血浆肾素活性和高血浆醛固酮水平。是继发性高血压的常见病因。该病多见于成年人。在高血压中发病率为3%~32%,平均为高血压患者的10%[1]。
作者 尹晓燕 张锦
出处 《山西医药杂志(上半月)》 CAS 2012年第5期486-488,共3页 Shanxi Medical Journal
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参考文献10

  • 1Mulatero P,Stowasser M,Loh KC,et al.Increased diagnosisof primary aldosteronism,including surgically correctableforms,in centers from five continents.J Clin EndoerinolMetab,2004,89(3):1045-1050.
  • 2KaPlan NM.The current epidemic of primry aldosteronism:causes and consequences.J Hyertension,2004,22:863-869.
  • 3Mosso L,Carvajal C,Gonzalez A,et al.Primary aldosteron-ism and hypertensive disease.Hypertension,2003,42(2):161-165.
  • 4Calhoun DA,Nishizaka MK,Zaman MA,et al.Hyperaldoste-ronism among black and white subjects with resistant hyper-tension.Hypertension,2002,40:892-896.
  • 5Montori VM,Young WF.Use of plasma aldosterone concen-tration-to-plasma rennin activity ratio as a screening test forprimary aldosteronism.A systematic review of the literature.Endocrinol Metab Clin North Am,2002,31:619-632.
  • 6Rossi E,Regolisti G,Negro A,et al.High prevalence of pri-mary aldosteronism using postcaptopril plasma aldosterone torennin ratio as a screening test among Italian hypertensives.Am J Hypertens,2002,15:896-902.
  • 7Mulatero P,Stowasser M,Loh KC,et al.Increased diagnosisof primary aldosteronism,including surgically correctableforms,in centers from five continents.J CIin EndocrinolMetab,2004,89:1045-1050.
  • 8Murayama T.Relationship between postoperation blood pres-sure change and renal pathophysiology in primary aldosteron-ism.Urol Int,1997,49:264-268.
  • 9杨建梅,郭晓蕙,董爱梅,田曙光.原发性醛固酮增多症的临床特点[J].临床荟萃,2005,20(24):1381-1384. 被引量:21
  • 10Stowasser M,Gordon RD.The aldosterone-renin ratio inscreening of prirmary a|dsoteronism.Endocirnologist,2004,14:267-276.

二级参考文献11

  • 1刘雪娜,周宪梁,刘亚欣,顾晴,陈改玲,孙晓昕,丰雷,惠汝太.原发性醛固酮增多症合并高血浆肾素活性的临床病例分析[J].中国分子心脏病学杂志,2004,4(4):233-234. 被引量:1
  • 2MULATERO P,STOWASSER M, LOH K C.Increased diagnosis of primary aldosteronism,including surgically correctable forms,in centers from five continents[J]. J Clin Endocrinol Metab,2004,89(3):1045-1050.
  • 3YOUNG W F Jr.Minireview:primary aldosteronism-changing concepts in diagnosis and treatment[J]. Endocrinology,2003,144(6):2208-2213.
  • 4STRUTHERS A D. Aldosterone-induced vasculopathy[J]. Molecular and Cellular Endocrinology,2004,217(1-2):239-241.
  • 5ROSSI G P, DI B V, GANZAROLI C, et al. Excess aldosterone is associated with alterations of myocardial texture in primary aldosteronism[J]. Hypertension,2002,40(1):23-27.
  • 6MILLIEZ P,GIRERD X,PLOUIN P F.Evidence for an increased rate of cardiovascular events in patients with primary aldosteronism[J]. J Am Coll Cardiol,2005,45(8):1243-1248.
  • 7STOWASSER M, GORDON R D. Primary aldosteronism-careful investigation is essential and rewarding[J]. Molecular and Cellular Endocrinology,2004,217(1-2):33-39.
  • 8STOWASSER M,GORDON R D,GUNASEKERA T G,et al. High rate of detection of primary aldosteronism, including surgically treatable forms, after ‘non-selective' screening of hypertensive patients[J]. J Hypertens,2003,21(11):2149-2157.
  • 9CONN J W, COHEN E L,ROVNER D R,et al. Normokalemic primary aldosteronism. A detectable cause of curable “essential” hypertension[J]. JAMA,1965,19(193):200-206.
  • 10ROSSI E, REGOLISTI G, NEGRO A, et al. High prevalence of primary aldosteronism using postcaptopril plasma aldosterone to renin ratio as a screening test among Italian hypertensives[J]. Am J Hypertens,2002,15(10 Pt 1):896-902.

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