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乳腺癌细胞可溶性FasL分泌及其对淋巴细胞凋亡的诱导 被引量:3

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出处 《肿瘤》 CAS CSCD 北大核心 2000年第4期310-312,共3页 Tumor
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  • 1Liu Q Y,Clin Cancer Res,1998年,4卷,1803页

同被引文献47

  • 1孙达,褚玉,楼岑,王乾君,占宏伟,何刚强.^(89)SrCl_2治疗转移性骨肿瘤的临床应用[J].中华肿瘤杂志,2005,27(8):499-501. 被引量:14
  • 2Shiraki K, Tsuji N, Shioda T, et al. Expression of Fas ligand in liver metastases of human colonic adenocarcinomas[J]. Proc Natl Acad Sci USA, 1997,94:6420.
  • 3Muller M, Strand S, Hug H, et al. Drug-induced apoptosis in hepatoma cells is mediated by the CD95 (APO-l/Fas) receptor/ligand system and involves activation of wild-type p53[J]. J Clin Invest, 1997,99:403.
  • 4Reybar U, Strater J, Kittstein W, et al. Colon carcinoma cell use different mechanism to escape CD95-mediated apoptosis[J]. Cancer Res, 1998,58:526.
  • 5Salo T, Irre S, Kitada S, et al. FAP-1: a protein lyrosine phosphalase that associates with Fas[J]. Science,1995,268:411.
  • 6Lee SH, Shin MS, Park WS, et al. Immunohistochemical localization of FAP-1, an inhibitor of Fas-mediated apoptosis, in normal and neoplastic human tissues[J]. APMIS, 1999,107:1101.
  • 7Ungefroren H, Voss M, Bernstorff WV, et al. Immunological escape mechanisms in pancreatic carcinoma [J].Ann Acad Sci, 1999,880:243.
  • 8Itakura M, Mort S, Park NH, et al. Both H PV and carcinogen contribute to the development of resistance to apoptosis during oral carcinogenesis[J]. Oncology, 2000,16: 591.
  • 9Leithauser F, Dhein J, Mechtersheimer G, et al. Constitutive and induced expression of APO-1, a new growth factor/tumor necrosis factor receptor superfamily, in normal and neoplastic cells[J]. Lab Invest, 1993, 69:415.
  • 10Bennett MW, O′connell J, L′sullivan GC, et al. The Fas counterattack in vivo: apoptotic depletion of tumor-infiltrating lymphocytes associated with Fas ligand expression by human esophageal carcinoma [J]. Immunotherapy,1998,48(160) :5669.

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