2Parinyasiri U, Ong - Ajyooth L, Parichati- kanond P, et al. Effect of fish oil on oxida- tive stress, lipid profile and renal function in IgA nephropathy [ J ]. J Med Assoc Thai,2004, 87 (2): 143-149.
3Walsh M, Sar A, Lee D, et al. Histopatho- logic features aid in predicting risk for progres- sion of IgA nephropathy [ J ]. Clin J Am Soc Nephrol, 2010, 5 (3): 425-430.
4Daha NA, Banda NK, Roos A, et al. Com- plement activation by ( auto - ) antibodies [J]. Md Immunol, 2011, 48 (14): 1656 - 1665.
5Cai GY, Chen XM. lmmunoglobulin A nephrop- athy in China: progress and challenges [ J ]. Am J Nephro1,2009,30(3 ) :268 - 273.
6Maeng YI, Kim MK, Park JB, et al. Glo- 1 .merular and tubular CAd depositions in IgA ne- phropathy:relations with histopathology andwith albuminuria [J]. Int J Clin Exp Pathol, 2013, 6 (5): 904-910.
7Nangaku M. Mechanisms of tubulointerstitial injury in the kidney: final common pathways to end - stage renal failure [ J ]. Intern Med, 2004, 43 (1): 9-17.
8Kostadinova - Kunovska S, Petrusevska G, Jovanovic R, et al. Histomorphometric anal- ysis of fibrosis in the renal interstitial com- partment [J]. Pfilozi, 2005, 26 (1): 51 -59.
9Rougier JP, Ronco P. Pathogenesis of im- mune glomerulonephritis[J]. Rev Prat, 2003, 53 (18): 1998-2004.
10Tam KY, Leung JC, Chan LY, et al. Mac- romolecular IgA1 taken from patients with fa- milial IgA nephropathy or their asymptomatic relatives have higher reactivity tomesangial cell.sin vitro [J]. Kidney Int, 2009, 75 (12): 1330-1339.
