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低温治疗的作用机制、应用范围及研究现状 被引量：5

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摘要 国际上将低温划分为轻度低温（mild hypothermia）33-35℃、中度低温（moderate hypothermia）28-32℃、深度低温（profound hypothermia）17-27℃和超深低温（ultraprofound hypothermia）2-16℃。江基尧教授于1993年首先将28-35℃轻中度低温统称为亚低温。
作者 孙昊 张劲松
机构地区 南京医科大学第一附属医院急诊科
出处 《中华急诊医学杂志》 CAS CSCD 北大核心 2012年第12期1403-1405,共3页 Chinese Journal of Emergency Medicine
关键词 低温治疗 应用 中度低温 轻度低温 深度低温 超深低温 亚低温
分类号 R651.15 [医药卫生—外科学]
  • 相关文献

参考文献42

  • 1Marion DW, Penrod LE, Kelsey SF, et al. Treatment of severe head injury with moderate hypothermia [ J]. N Engl J Med, 1997, 336 (8) : 540-546.
  • 2Jiang JY, Yu MK, Zhu C. Effect of long-term mild hypothermia on patients with severe traumatic brain injury. 1 year follow up of 87 case [J]. J Neurosurg, 2000, 93 (4): 546-549.
  • 3曾红科,邓医宇.心肺复苏后亚低温脑保护治疗[J].中华急诊医学杂志,2011,20(12):1353-1354. 被引量：12
  • 4李恒,杨正飞,符岳,朱军,姜骏,余涛,方向韶,黄子通.缺血期亚低温保存再灌注后心肌细胞收缩功能的研究[J].中华急诊医学杂志,2011,20(11):1143-1148. 被引量：4
  • 5Polderman KH. Mechanisms of action, physiological effects, and complications of hypothermia [ J ]. Crit Care Med, 2009, ( 7 Suppl) : S186-202.
  • 6Liou AK, Clark RS, Henshall DC, et al. To die or not to die for neurons in ischemia, traumatic brain injury and epilepsy: a review on the stress-activated signaling pathways and apoptotic pathways [J]. Prog Neurobiol, 2003, 69 (1) : 103-142.
  • 7Baker AJ, Zornow MH, Grafe MR, et al. Hypothermia prevents ischemia-induced increases in hippocampal glyeine concentrations in rabbits [J]. Stroke, 1991, 22 (5) : 666-673.
  • 8Globus MY, Alonso O, Dietrich WD, et al. Glutamate release and free radical production following brain injury: effects of post traumatic hypothermia [J]. J Neurochem, 1995, 65 (4) : 1704- 1711.
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  • 10Chi OZ, Liu X, Weiss HR. Effects of mild hypothermia on blood- brain barrier disruption during isoflurane or pentobarbital anesthesia [J]. Anesthesiology, 2001, 95 (4): 933-938.

二级参考文献14

  • 1杨兴易.心肺复苏研究的几个热点问题[J].第二军医大学学报,2004,25(11):1161-1163. 被引量：12
  • 2Gazmuri R J, Weil MH, Bisera J, et al. Myocardial dysfunction after successful resuscitation from cardiac arrest [ J ]. Crit Care Med, 1996, 24 (6):992-1000.
  • 3Hsu CY, Huang CH, Chang WT, et al. Cardioprotective effect of therapeutic hypothermia for postresuscitation myocardial dysfunction [J]. Shock, 2009, 32 (2):210-216.
  • 4Fu JJ, Gao J, Pi RB, et al. An optimized protocol for culture of cardiomyocyte from neonatal rat [ J ]. Cytotechnology, 2005, 49 (6) : 109-116.
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  • 6Mizukami Y, Iwamatsu A, Aki T, et al. ERK1/2 regulates intracellular ATP levels through enolase expression in cardiomyocytes exposed to ischemic hypoxia and reoxygenation [J]. J Biol Chem, 2004, 279 (48) : 50120-50131.
  • 7Ristagno G, Tantillo S, Sun S J, et al. Hypothermia improvesventricular myocyte contractility under conditions of normal perfusion and after an interval of ischemia [ J]. Resuscitation, 2010, 81 (7) : 898-903.
  • 8Yu T, Barbut D, Ristagno G, et al. Survival and neurological outcomes after nasopharyngeal cooling or peripheral vein cold saline infusion initiated during CPR in a porcine model of prolonged cardiac arrest [J]. Crit Care Med, 2010, 38 (3) : 916-921.
  • 9Ozean C, Bienengraeber M, Dzeja PP, et al. A Potassium channel openers protect cardiac mitochondria by attenuating oxidant stress atreoxygenation [ J ]. Am J Physiol Heart Circ Physiol, 2002, 282 (2) : H531-H539.
  • 10Ali SS, Marcondes MC, Bajova H, et al. Metabolic depression and increased reactive oxygen species production by isolated mitochondria at moderately lower temperatures [ J ]. J Biol Chem, 2010, 285 (42) : 32522-32528.

共引文献52

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中华急诊医学杂志
2012年 第12期

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