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缺血性脑卒中信号转导通路研究进展 被引量:10

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摘要 短暂或持久的局灶性脑缺血可引起一系列病理生理变化而导致脑损伤,且随时问和缺血程度增加而加重。由此引发的缺血性脑卒中,其发病机制复杂,能量代谢障碍、兴奋性氨基酸毒性作用、炎性反应、半暗带去极化及细胞凋亡等共同参与了其病理生理变化过程。
出处 《中华老年心脑血管病杂志》 CAS 北大核心 2013年第2期218-220,共3页 Chinese Journal of Geriatric Heart,Brain and Vessel Diseases
基金 院级基金资助项目(201104)
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参考文献25

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二级参考文献51

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