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遗传性多发性骨软骨瘤并膝外翻矫形术后并发腓总神经损伤一例 被引量:2

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摘要 笔者于2011年4月治疗遗传性多发骨软骨瘤并膝外翻截骨矫形术后并发腓总神经损伤1例,报告如下。1病例报告,患者,女。16岁,因“左膝关节渐进性外翻畸形伴跛行3年余”人院。患者家族中有遗传性多发性骨软骨瘤病史。查体:跛行步态,双下肢等长(约72em),内踝间距7cm。左下肢轻度内旋畸形,左膝关节外翻18°,前屈(胫骨近端向前成角)250畸形,左胫骨前肌、拇趾背伸肌、腓骨长短肌肌力5级。
出处 《中国骨与关节损伤杂志》 2013年第2期121-121,共1页 Chinese Journal of Bone and Joint Injury
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  • 1李文锋,侯树勋,王庆雷.微创截骨外固定器固定治疗膝内翻畸形[J].中国矫形外科杂志,2006,14(7):512-513. 被引量:1
  • 2朱盛修 王惠敏 等.外固定不当致周围神经伤28例分析[J].中华外科杂志,1988,26(5):344-344.
  • 3[4]Fabre T,Piton C,Andre D,et al.Petroneal nerve entrapment[J].J Bone Joint Surg Am,1998,80 (1):47~53
  • 4[5][美]傅兰谷著,过邦辅编译.临床骨科生物力学基础[M].上海:上海远东出版社,1993.112~116
  • 5Voloc A, Esterle L, Nguyen TM, et al. High prevalence of genu varum/valgum in European children with low vitamin D status and insufficient dairy products/calcium intakes. Eur J Endocrinol, 2010, 163(5): 811-817.
  • 6Haddad FS, Bentley G. Total knee arthroplasty after high tibial osteotomy: a medium-term review. J Arthroplasty, 2000, 15(5):597-603.
  • 7Wyss TF, Schuster AJ, Munger P, et al. Does total knee joint replacement with the soft tissue balancing surgical technique maintain the natural joint line? Arch Orthop Trauma Surg, 2006, 126(7): 480-486.
  • 8Yan CH, Chiu KY, Ng FY. Total knee arthroplasty for primary knee osteoarthritis: changing pattern over the past 10 years. Hong Kong Med J, 2011, 17(1): 20-25.
  • 9Waciakowski D, Urban K, Karpao K. Valgus high tibial osteotomy - long-term results. Acta Chir Orthop Traumatol Cech, 2011, 78(3): 225-231.
  • 10Insall JN, Dorr LD, Scott RD, et al. Rationale of the Knee Society clinical rating system. Clin Orthop Relat Res, 1989(248): 13-14.

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  • 1刘振江,张立军,马瑞雪,刘卫东.髂骨植骨尺骨延长治疗多发性骨软骨瘤前臂畸形的疗效评价[J].中国矫形外科杂志,2005,13(13):990-992. 被引量:16
  • 2郭世炳,冯卫.遗传性多发性骨软骨瘤[J].中国医师进修杂志(外科版),2007,30(4):72-74. 被引量:3
  • 3Garner OB, Yamaguchi Y, Esko JD, et al. Small changes in lymphocyte development and activation in mice through tissue specificalteration ofheparan sulphate[J]. Immunology, 2008, 125(3): 420-429.
  • 4Chen S, Wassenhove-Mccarthy D J, Yamaguchi Y, et al. Loss of heparan sulfate glycosaminoglyean assembly in podocytes does not lead to proteinuria[J]. Kidney Int, 2008, 74(3): 289-299.
  • 5Iwao K, Inatani M, Mastsumoto Y, et al. Heparan sulfate deficiency leads to Peters anomaly in mice by disturbing neural crest TGF-beta2signaling[J]. J Clin Invest, 2009, 119(7): 1997-2008.
  • 6Iwao K, Inatani M, Ogata-Iwao M, et al. Heparan sulfate deficiency in periocular mesenchyme causes microphthalmia and ciliary body dysgenesis[J]. Exp Eye Res, 2010, 90(1): 81-88.
  • 7Kucharzewska P, Welch JE, Birgersson JA. Establishment of heparan sulphate deficient primary endothelial cells from EXT- l(flox/flox) mouse lungs and sprouting aortas[J]. In Vitro Cell Dev Biol Anim, 2010, 46(7): 577-584.
  • 8Bishop JR, Schuksz M, Esko JD. Heparan sulphate proteoglycans fine-tune mammalian physiology[J]. Nature, 2007, 446(7139): 1030-1037.
  • 9李剑雄,周瑞方.多发性骨软骨瘤l例[J].长江大学学报·自然科学版医学卷,2010,7(3):198-199.
  • 10Roach JW, Klatt JW, Faulkner ND. Involvement of the spine in patients with multiple hereditary exostoses[J]. J Bone Joint Surg Am, 2009, 91A(8): 1942-1948.

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