2Zhang Y,Widmayer MA,Zhang B,et al.Suppression of post—ischemic-induced FOS protein expression by an antisense oligonucleotide to c-fos mRNA leads to increased tissue damage.Brain Res,1999,832:112.
3Tchelingerian IL Saux FL,Pouzet B,et al.Widespread neuronal expression of c—fos throughout the brain and local expression in glia following a hippocampal injury.Neuroscience Letters,1997,226:175.
4Macara IG. Oncogenes and celluar signal transduction. Physiol Rev,1989, 69:797.
5Jorgensen MB. Delayed c-fos proto-oncogene expression in the rat hippocampus induced by transient global ischemia: in situ hybridization study. Brain Res, 1989, 484:393.
6Curan T. The fos oncogene. In: Reddy EP, et al(eds). The oncogene handbook. Amsterdam: Elserer Press, 1988. 307.
7Koizumi J, Yoshida Y, Nakazawa T, et al. Experimental studies of ischemic brain edema, Ⅰ: A new experimental model of cerebral embolism in rats in which recirculation can be introduced in the ischemic area. Jpn J Stroke, 1986, 8:1.
8Nagasawa H, Kogure K. Correlation between cerebral blood flow and histologic changes in a new rat model of middle cerebral artery occlusion. Stroke, 1989, 20(8): 1037.
9Onodera H, Kogure K, Ono Y, et al. Proto-oncogene c-fos is transiently induced in the rat cerebral cortex after forebrain ischemia.Neurosci Lett, 1989, 98:101.
10Popovici T, Represa A, Crepel V, et al. Effects of kainic acid-induced seizure and ischemia on c-fos-like proteins in rat brain. Brain Res, 1990, 536:183.
