2[1]Jackman WM,Beckman K J,McClelland JH,et al.Lazzara R.Treatment of supraventricular tachycardia due to atrioventricular nodal reentry by radiofrequency catheter ablation of slow-pathway conduction.N Engl J Med,1992,327:313-318.
3[2]Haissaguerre M,Gaita F,Fischer B,et al.Elimination of atrioventricular nodal reentrant tachycardia using discrete slow potentials to guide application of radiofrequency energy.Circulation,1992,85:2162-2174.
4[3]de Bakker JMT,Lob P,Hocini M,et al.Double component action potentials in the posterior approach to the atrioventricular node:do they reflect activation delay in the slow pathway.? J Am Coll Cardiol,1999,34:570-577.
5[4]McGuire MA,de Bakker JMT,Vermeulen JT,et al.Origin and significance of double potentials near the atrioventricular node:correlation of extracellular potentials,intracellular potentials,and histology.Circulation,1994,89:2351-2360.
6[7]Trohman RG,Pinski SL,Sterba R,et al.Evolving concepts in radiofrequency catheter ablation of atrioventricular nodal reentry tachycardia.Am Heart J,1994,128:586 -595.
7[8]Lin JL,Lin FY,Lo HM,et al.Perinodal slow potential as a local guid for transcatheter radiofrequency ablation of atrioventricular nodal reentrant tachycardia:therapeutic efficacy and electrophysiologicalmechanisms of success.Br Heart J,1995,74:268-276.
