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偏侧舞蹈症病因研究进展 被引量:5

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摘要 偏侧舞蹈症是因锥体外系病变而导致的一组临床综合征,临床多表现为一侧肢体或单纯上、下肢在发生偏瘫后迅速或间隔一段时间出现快速、无目的、粗大的、不规则、不自主的舞蹈样动作,以肢体近端为主,面部舞蹈样动作多表现为皱额、挤眼、努嘴、吐舌、口角牵动、下颌摇动等。舞蹈样运动多以上肢表现重而下肢和面部相对较轻,情绪紧张时较明显,而安静时减轻、睡眠时消失。
出处 《中风与神经疾病杂志》 CAS CSCD 北大核心 2013年第6期573-574,共2页 Journal of Apoplexy and Nervous Diseases
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参考文献18

  • 1胡维铭;王维治.神经内科主治医师1000问[M]北京:中国协和医科大学出版社,2007584.
  • 2黄如训;梁秀玲;刘焯霖.临床神经病学[M]北京:人民卫生出版社,1996.
  • 3Rice ME,Perez-Pinzon MA,Lee EJK. Ascorbic acid,but not glutathione,is taken up by brain slices and preserves cellmorphology[J].Journal of Neurophysiology,1994.1591-1596.
  • 4白玉海,曹丽华,崔志堂,张锦,佟晓艳,曹淑梅,张德梅.急性脑血管病致偏侧舞蹈症的临床分析[J].中国基层医药,2006,13(3):496-496. 被引量:2
  • 5Qi X,Yan YY,Gao Y. Hemichorea associated with non-ketotic hyperglycaemia:A case report[J].Diabetes Research and Clinical Practice,2012,(01):1-3.
  • 6Lai PH,Tien RD,Chang MH. Chorea-ballismus with nonketotic hyperglycemia in primary diabetes mellitus[J].American Journal of Neuroradiology,1996.1057-1064.
  • 7Shah DE,Ho DM,Chang C. Hemiehorea-hemiballism:an explanation for MR signal changes[J].American Journal of Neuroradiology,1998.863-870.
  • 8Ohara S,Nakagawa S,Tabata K. Hemiballism with hyperglycemia and striatal T1-MRI hyperintensity:an autopsy report[J].Movement Disorders,2001,(3):521-525.
  • 9Nath J,Jambhekar K,Rao C. Radiological and pathological changes in hemiballism-hemiehorea with striatal hyperintensity[J].Journal of Magnetic Resonance Imaging,2006.564-568.
  • 10Hsu JL,Wang HC,Hsu WC. Hypergiycemia-induced unilateral basal ganglion lesions with and without hemichorea A PET study[J].Journal of Neurology,2004.1486-1490.

二级参考文献25

  • 1杨雪,贺茂林,刘利新,吴逊.急性脑血管病中的同侧舞蹈症和舞动症[J].中华神经科杂志,1996,29(4):222-224. 被引量:24
  • 2母春华,卢登茜,郭书英,母成贤.脑梗塞所致半侧舞蹈病(附10例报告)[J].河北医科大学学报,1996,17(5):282-283. 被引量:9
  • 3林晓东.老年人卒中性偏侧舞蹈症[J].中华老年医学杂志,1993,12(5):303-303.
  • 4Piccolo I, Defanti CA, Solivefi P, et al. Cause and course in a series of patients with sporadic chorea. J Neurol,2003,250:429-435.
  • 5Hoppensteadt DA, Fabbrini N, Bick RL, et al. Laboratory evaluation of the antiphospholipid syndrome [ J ]. Hematol Oncol Clin North Am, 2008 ;22 ( 1 ) : 19-32.
  • 6Wilson WA, Gharavi AE, Koike T, et al. International consensus state- ment on preliminary classification criteria for definite antiphospholipid syndrome: report of art international workshop [J]. Arthritis Rheum, 1999 ;42 : 1309-11.
  • 7Miyakis S, Lockshin MD, Atsumi T, et al. International consensus state- ment on an update of the classification criteria for definite antiphospholip- id syndrome(APS) [ J]. J Thromb Haemost,2006 ;4 ( 2 ) :295-306.
  • 8Espinosa G, Cervera R, Font J, et al. Antiphospholipid syndrome : patho- genic mechanisms [ J ]. Autoimmun Rev,2003 ;2 ( 1 ) :86-93.
  • 9Krause I, Leibovici L, Blank M, et al. Clusters of disease manifestations in patients with antiphospholipid syndrome demonstrated by factor analysis [ J ]. Lupus,2007 ; 16 (3) : 176-80.
  • 10Cuadrado MJ, Khamashta MA, Ballesteros A, et al. Can neurologic mani- festations of Hughes (antiphospholipid) syndrome be distinguished from multiple sclerosis? Analysis of 27 patients and review of the literature ( J ). Medicine(Baltimore) ,2000 ;79:57-68.

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