摘要
本文进一步证明草酸能显著诱导黄瓜对炭疽病的系统抗性 ,同时发现硫酸亚铁亦具有显著的诱抗效果。草酸能诱导局部叶片及系统叶片中壁共价键结合态、壁离子键结合态和可溶态过氧化物酶 ( POD)活性提高 ,其中离子键结合态 POD活性提高量最大 ,共价键结合态 POD活性提高倍数最大。草酸处理后还原型抗坏血酸 ( As A)与氧化型抗坏血酸 ( DHAs A)的比值和亚铁离子 ( Fe2 +)与三价铁离子 ( Fe3+)的比值均明显增加。对壁结合态 POD、Fe2 +、As A在草酸诱导抗病性中的作用进行了较深入的讨论。
The study showed that oxalate-induced systemic resistance to anthracnose in cucumber leaves was due to the function of oxalate anion group. It was meanwhile observed that FeSO 4 was also a good inducer to increase the systemic resistance. The activity of different forms of peroxidase (POD), including soluble, ionically cell wall bound and covalently cell wall bound peroxidase, was significantly induced by oxalate. The ratio of ascorbic acid (AsA) to dehydroascorbic acid (DHAsA) and ferrous (Fe 2+) to ferric (Fe 3+) iron was remarkably increased by oxalate treatment. The significance of cell wall bound peroxidases, ferrous iron and ascorbate in oxalate-induced resistance was disccussed.
出处
《植物病理学报》
CAS
CSCD
北大核心
2000年第2期153-157,共5页
Acta Phytopathologica Sinica
基金
霍英东基金! (0 5 0 10 2 1)
广东省自然科学基金! (940 377)
关键词
黄瓜
草酸
炭疽病
过氧化物酶
抗坏血酸
cucumber
oxalate
anthracnose
peroxidase
ascorbate
ferrous iron
