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氯吡格雷抵抗研究的新分歧 被引量:5

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摘要 双联抗血小板治疗是急性冠状动脉综合征(ACS)和经皮冠状动脉介入(PCI)的标准化治疗[1]。但尽管双重抗血小板治疗,仍有患者发生心血管事件,最严重的是支架内血栓形成[2]。最近新发现氯吡格雷抵抗是PCI术中无血流的机制之一[3],血栓事件发生后的病死率为15%~45%,再梗死率为60%~70%。
出处 《国际检验医学杂志》 CAS 2013年第15期1990-1992,共3页 International Journal of Laboratory Medicine
基金 甘肃省卫生行业科研计划项目(2010-652)
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参考文献31

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二级参考文献52

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共引文献27

同被引文献88

  • 1苏冠华,王朝晖.血小板膜GPⅡb/Ⅲa基因多态性与冠心病[J].心血管病学进展,2006,27(z1):14-16. 被引量:3
  • 2郝盼盼,陈玉国,张运.氯吡格雷抵抗相关因素的研究进展[J].心血管病学进展,2009,30(6):991-994. 被引量:21
  • 3吴小利,李健,王成彬.抗血小板药物新进展[J].临床检验杂志(电子版),2012,1(4):221-227. 被引量:1
  • 4张永珍,高炜.氯吡格雷抵抗的机制与对策[J].中华内科杂志,2006,45(3):250-252. 被引量:12
  • 5黄震华.氯吡格雷抵抗[J].中国新药与临床杂志,2007,26(9):695-698. 被引量:7
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