1Silverberg SG, Mutter GL, Kurman R J, et al. Tumors of the uterine corpus:epithelial tumors and related lesions. In: Tavassoli FA, Stratton MR, editors. WHO classification oftumors : pathology and genetics of tumors of the breast and female genital organs. Lyon : IARC Press, 2003:221-232.
2Jia L, Liu Y, Yi X,et al. Endometrial glandular dysplasia with frequent p53 gene mutation:a genetic evidence sup- porting its precancer nature for endometrial serous carcino- ma[ J ] Clin Cancer Res,2008,14 (8) :2263-2269.
3Soltysik K, Czekaj P. Membrane estrogen receptors-is it an alternative way of estrogen action? [ J]J Physiol Pharma- col,2013,64(2) :129-142.
4Wik E,Rader MB,Krakstad C, et al. Lack of estrogen re- ceptor-c is associated with epithelial-mesenchymal transi- tion and PI3K alterations inendometrial carcinoma [ J] Clin Cancer Res, 2013,19 (5) : 1094-1105.
5Nishijima Y, Yoshida T, Nakamura K, et al. hMLH1 pro- moter methylation status causes different expression pat- terns of estrogen receptor protein with endometrial lesion progression[ J]. Rinsho Byori,2013,61(2):97-103.
6Guo T, Li B, Gu C. Expression of hPEBP4 negatively cor- relates with estrogen and progesterone receptors in endom- etrial carcinoma [ J ]. J BUON, 2013,18 ( 2 ) : 465-470.
7Wei Y, Zhang Z, Liao H, et al. Nuclear estrogen receptor- mediated Notch signaling and GPR30-mediated PI3K/ AKT signaling in the regulation of endometrial cancer cell proliferation [ J ]. Oncol Rep,2012,27 (2) :504-510.
8Ge X, Guo R, Qiao Y, et al. The G protein-coupled recep- tor GPR30 mediates the nontranscriptional effect of estro- gen on the activation of PI3K/Akt pathway in endometrial cancer cells [ J ]. Int J Gynecol Cancer, 2013,23 ( 1 ) : 52- 59.
9Mhawech-Fauceglia P, Wang D, Samrao D, et al. Trefoil factor family 3 ( TFF3 ) expression and its interaction with estrogen receptor (ER) in endometrial adenocarcinoma [ J]. Gynecol Oneol,2013,130( 1 ) : 174-180.
10Zhou C, Steplowski TA, Dickens HK, et al. Estrogen in- duction of telomerase activity through regulation of the mitogen-activated protein kinase (MAPK) dependent pathway in human endometrial cancer cells [ J ]. PLoS One,2013,8(2) :e55730.
