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阿尔茨海默病与微循环 被引量:3

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作者 刘声远
出处 《微循环学杂志》 2000年第3期11-13,共3页 Chinese Journal of Microcirculation
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  • 1Tanzi RE, Koracs EM, Kim TW, et al. The gene defects responsible for familial Alzheimer's disease. Neurobiol Dis, 1996, 3(3):159~168.
  • 2Johnson-Wood K, Lee M, Motter R, et al. Amyloid precursor protein processing and A beta42 deposition in a transgenic mouse model of Alzheimer disease. Proc Nati Acad Sci, 1997, 94:1 550~1 555.
  • 3Tilvis RS, Strandberg TE, Jaua K, et al. Apolipoprotein E phenotypes, dementia and mortality in a prospective population sample. J Am Geriatr Soc, 1998, 46:712~715.
  • 4Citron M, Westway D, Xu W, et al. Mutant presenilins of Alzheimer's disease increase production of 42-residue amyloid beta-protein in both transfected cells and transgenic mice. Nat Med, 1997, 3:67~72.
  • 5Chen M, Fernandez HL. The Alzheimer's plaques, tangles and memory deficits may have a common origin. Part Ⅳ: can calpain act as alpha-secretase? Front Biosci, 1998, 3:A27~A31.
  • 6Smale G, Nichols NR, Brady DR, et al. Evidence for apoltotic cell death in Alzheimer's disease. Exp Neurol, 1995, 133:225~230.

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