1Wickelgren I.First components found for new kidney filter[J].Science,1999,286(5438):225 - 226.
2Shih NY, Li J, Karpitskii V, et al. Congenital nephritic syndrome in mice lacking CD2 associated protein[J].Science, 1999, 286(5438):312-315.
3Kawachi H, Abraharson DR, St John PL, et al. Developmental expression of the nephritogenic antigen of monoclonal antibody 5-1-6[J]. Am J Pathol, 1995,147(3):823-833.
4Kestila M, Mannikko M, Holmberg C, et al. Congenital nephritic syndrome of the Finnish type maps to the long arm of chromosome 19[J]. Am J Hum Genet, 1994,54(5):757-764.
5Mannikko M, Kestaila M, Holmberg C, et al. Fine mapping and happlotype analysis of the locus for congenital nephritic syndrome on chromosome 19q13.1[J]. Am J Hum Genet, 1995,57(6):1377- 1383.
6Kestila M, Lenkkeri U, Mannikko M, et al. Positionally cloned gene for a novel glomerular protein-nephrin-is mutated in congenital nephrotic syndrome[J].Mol Cell, 1998,1(4):575- 582.
7Lenkkeri U, Marmikko M, McCready P, et al. Structure of the gene for congenital nephrotic syndrome of the Finnish type(NPHS1)and characterization of mutations[J]. Am J Hum Genet, 1999, 64(1):51-61.
8Ruotsalainen V, Ljungberg P, Wartiovaara J, et al. Nephrin is specifically located at the slit diaphragm of glomerular podocytes[J]. Proc Natl Acad Sci USA, 1999,96(7):7962-7967.
9Tryggvason K. Unraveling the mechanisms of glomerular ultrafiltration;nephrin, a key component of the slit diaphragm[J]. J Am Soc Nephrool. 1999,10(11):2440-2445.
10Furness PN, Hall LL, Shaw JA, et al. Glomerular expression of nephrin is decreased in acquired human nephritic syndrome[J].Nephrol Dial Transplant, 1999,14(12):1234- 1237.
4Mundel P, Heid HW, Mundel TM, et al. Synaptopodin: an actin - associated protein in telencephalic dendrites and renal podo- cytes. J Cell Biol, 1997,139 ( 1 ) : 193 - 204.
5Churg J, Bemstein J, Glassock RJ. Renal disease - Classification and at las of glomerula disease, second edition. New York: Igaku - shoin Medical Publishs Inc, 1995.4 - 5.
6Kerjaschki D. Dysfunction of cell biological mechanisms of -is- ceral epithelial ceils(podocytes) in glomerular diseases. Kidne- sy Int,1994,45(2) :300 -313.
7Kriz W, Gretz KV. Lemley : Progression of glomerular diseases : is the podocyte the culprit. Kidney Int, 1998,54 (3) :687 -697.
8Pagtalunan MPL, Miller S, Jumping Eagle, et al. Podoeyte loss and progressi-e glomerular injury in type 11 diabetes. J Clin In- -est, 1997,99 (2) :342 - 348.
9Barisoni L, Kriz W, Mundel P, et al. The dysregulated podocyte phenotype : a no-el concept in the pathogenesis of collapsing idi- opathic focalsegmental glomerulosclerosis and HIV - associated nephropathy. J the Amer Soci Nephrol, 1999,10 ( 1 ) : 51 - 61.
10Kemeny E, Durmuller U, Nickeleit V, et al. Distribution of podo- cyte protein (44 KD) in different types of glomerular diseases. Virchows Arch, 1997,431 (3) :425 - 430.
