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慢性淋巴细胞白血病合并真性红细胞增多症一例JAK2V617F突变研究 被引量：2

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摘要 1病例报告 患者男，85岁。因“体柃发现血细胞增多2个月余”于2010-02—06入住杭州市第一人民医院血液科。
作者 谢亚萍 黄细莲 陈况 高大泉 刘利蓉 钱申贤
机构地区 杭州市第一人民医院血液科
出处 《中华肿瘤防治杂志》 CAS 北大核心 2014年第3期232-233,236,共3页 Chinese Journal of Cancer Prevention and Treatment
关键词 真性红细胞增多症 JAK2 慢性淋巴细胞白血病 骨髓增殖性肿瘤
分类号 R733.7 [医药卫生—肿瘤]
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参考文献16

  • 1Campbell PJ,Green AR. The myeloproliferative disorders[J].{H}New England Journal of Medicine,2006,(23):2452-2466.
  • 2Kikushige Y,Ishikawa F,Miyamoto T. Self-renewing hematopoietic stem cell is the primary target in pathogenesis of human chronic lymphocytic leukemia[J].{H}CANCER CELLS,2011,(02):246-259.
  • 3Weigert O,Weinstock DM. The evolving contribution of hematopoietic progenitor cells to lymphomagenesis[J].{H}Blood,2012,(13):2553-2561.
  • 4Laurenti L,Tarnani M,Nichele I. The coexistence of chronic lymphocytic leukemia and myeloproliferative neoplasms:a retrospective multicentric GINEMA experience[J].{H}American Journal of Hematology,2011,(12):1007-1012.
  • 5Swierczek S,Nausova J,Jelinek J. Concomitant JAK2 V617Fpositive polycythemia vera and B-cell chronic lymphocytic leukemia in three patientsoriginating from two separate hematopoietic stem cells[J].{H}American Journal of Hematology,2013,(02):157-158.
  • 6Zehentner BK,Loken MR,Wells DA. Jak2 mutation can occur exclusively in the erythroid lineage and be absent in granulocytes and progenitor cells in classic myeloproliferative disorders[J].{H}American Journal of Hematology,2006,(10):806-807.
  • 7牛志云,郭晓玲,张敬宇,杨琳,罗建民.CMPN基因突变及其机制与生物学意义的探讨[J].中华肿瘤防治杂志,2013,20(15):1175-1178. 被引量：1
  • 8James C,Ugo V,Le Couédic JP. A unique clonal JAK2 mutation leading to constitutive signaling causes polycythaemia vera[J].{H}NATURE,2005,(7037):1144-1148.
  • 9Baxter EJ,Scott LM,Campbell PJ. Acquired mutation of the tyrosine kinase JAK2 in human myeloproliferative disorders[J].{H}LANCET,2005,(9484):1054-1061.
  • 10Henry L,Carillo S,Jourdan E. Association of essential thrombocythemia and chronic lymphocytic leukemia:absence of the V617F JAK2 mutation in the lymphoid compartment[J].{H}American Journal of Hematology,2007,(06):500-501.

二级参考文献13

  • 1James C,Ugo V,Le Couedic JP,et al. A unique clonal JAK2 mu-tation leading to constitutive signalling causes polycythaemiavera[J]. Nature,2005 ,434(7037) : 1144-1148.
  • 2Kannim S,Thongnoppakhun W, Auewarakul CU. Two-round al-lele specific-polymerase chain reaction:a simple and highly sensi-tive method for JAK2-V617F mutation detection[J]. Clin ChimActa,2009,401(1-2);148-151.
  • 3Levine RL ? W alleigh M, Cools J ? et al. Activating mutation in thetyrosine kinase JAK2 in polycythmia vera,essential thrombo-cythmi-a,and myeloid metaplasia with myelofibrosis[J]. CancerCell,2005,7(4) :387-397.
  • 4Laubach JP,Fu P, Jiang XH,et al. Polycythemia vera erythroidprecursors exhibit increased proliferation and apoptosis resist-ance associated with abnormal RAS and PI3K pathway activation[J]. Exp Hematol,2009,37(12) :1411-1422.
  • 5Panani AD. Janus kinase 2 mutations in Phladelphia negative chronicmyeloproliferat ive disorders:climcal implications[J]. Cancer Letters,2009,284(1):7-14.
  • 6Oku S, Takenaka K, Kuriyama T,et al. JAK2V617F uses distinctsignalling pathways to induce cell proliferation and neutrophil activa-tion[J]. Br J Haematol,2010,150(3) :334-344.
  • 7Chen CC,Stairs DB,Boxer RB. Autocrine prolactin induced bythe Pten-Akt pathway is required for lactation initiation and pro-vides a direct link between the Akt and Stat5 pathways [J].Genes Dev,2012,26(19) :2154-2168.
  • 8Tyler RK’Shpiro N,Marquez R,et al. VX-680 inhibits AuroraA and Aurora B kinase activity in human cells[J], Cell Cycle?2007,6(22):2846-2854.
  • 9Muller P,Boutros M, Zeidler MP. Identification of JAK/STATpathway regulators-insights from RNAi screens. Identifica-tion of JAK/STAT pathway regulators-insights from RNAiscreens[J]. Ann Biol Clin (Paris) ,2012,70(5):591-594.
  • 10路秀英,李晓明,王俊阁.JAK2激酶抑制剂AG490抑制喉鳞癌细胞侵袭力的实验[J].肿瘤防治研究,2008,35(9):664-664. 被引量：2

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中华肿瘤防治杂志
2014年 第3期

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