期刊文献+

高脂血症和骨质疏松关系的临床和基础研究进展 被引量:2

下载PDF
导出
摘要 骨质疏松(osteoporosis,OP)是一种以骨质减少,骨的微观结构退化为特征,致使骨的脆性增加以及易于发生骨折的一种全身性骨骼疾病。OP好发于65岁以上老年人群,
作者 吴萌萌 金贤
出处 《老年医学与保健》 CAS 2014年第1期58-62,共5页 Geriatrics & Health Care
  • 相关文献

参考文献26

  • 1Reginster JY, Burlet N. Osteoporosis: a still increasing prevalence [J]. Bone, 2006, 38 (2 suppl 1): $4-$9.
  • 2Bilezikian JP, Davidson M, Hendrix S, et aL Co-morbidity of decreased bone mineral density (BMD) and increased chol- esterol levels among women aged 65 years and older: results from NHANESIII [J]. J Bone Miner Res, 2005,201 (9): $219.
  • 3Hsu YH, Vermers SA, Terwedow HA, et al. Relation of body composition, fat mass, and serum lipids to osteoporotic fractures and bone mineral density in Chinese men and women [J]. Am J Clin Nutr, 2006, 83 (1): 146-154.
  • 4Cui LH, Shin MH, Chung EK, et al. Association between bone mineral densities and serum lipid profiles of pre- and post- menopausal rural women in South Korea [J]. Osteoporosis Int, 2005, 16 (12): 1975-1981.
  • 5Adami S, Braga V, Zamboni M, et al. Relationship between lipids and bone mass in 2 cohorts of healthy women and men [J]. CalcifTissuelnt, 2004, 74 (2): 136-142.
  • 6Pliatsika P, Antoniou A, AlexandrouA, et al. Serum lipid levels and bone mineral density in Greek postmenopausal wom- en [J]. GynecolEndocrinol, 2012,28 (8): 655-660.
  • 7Parhami F,Garfinkel A, Demer LL. Role of lipids inosteoporosis.Arterioscler Thromb Vasc Biol [J]. 2000, 20 (11): 2346-2348.
  • 8Parhami F, Morrow AD, Balucan J, et al. Lipid Oxidation Products Have Opposite Effects on Calcifying Vascular Cell and Bone Cell Differentiation : A Possible Explanation tbr the Para- dox of Arterial Calcification in Osteoporotic Patients [J]. Ar- terioscler Thromb Vasc Biol, 1997, 17 (4): 680-687.
  • 9Brodeur MR, Brissette L, FalstraultL, et al. Influence of oxidizedlow-densitylipoproteins (LDL) on the viability of os- teoblastic cells [J]. Free Radic Biol Med, 2008 , 44 (4): 506-517.
  • 10Valenti MT, Garbin U, Pasini A, et al. Role of Ox-PAPCs in the Differentiation of Mesenchymal Stem Cells (MSCs) and Runx2 and PPAR2,/Expression in MSCs-Like of Osteoporotic Patients [J]. PLoS One, 2011, 6 (6): e20363.

同被引文献41

  • 1张咏梅,林华.冠心病和骨质疏松[J].中国骨质疏松杂志,2005,11(2):266-268. 被引量:23
  • 2张咏梅,林华,王春.高血压冠心病与骨质疏松骨量的相关性研究[J].实用老年医学,2006,20(2):116-117. 被引量:22
  • 3Mazziotti G,Bilezikian J, Canalis E, et al. New understanding and treatments for osteoporosis[J]. Endoerine,2012,41(1):58-69.
  • 4Cagnetta V, Patella V. The role of the immune system in the physiopathology of osteoporosis [J]. Clin Cases Miner Bone Metab,2012,9(2) :85-88.
  • 5Nakashima Y, Haneji T. Stimulation of osteoclast formation by RANKL requires interferon regulatory factor-4 and is inhibited by simvastatin in a mouse model of bone loss[J]. PIoS One, 2013,8 (9) : e72033.
  • 6Ruan F, Zheng Q, Wang J. Mechanisms of bone anabolism regula- ted by statins[J]. Biosei Rep,2012,32(6):511-519.
  • 7Lissenberg-Thunnissen SN, de Gorter DJ, Sier CF, et al. Use and efficacy of bone morphogenetic proteins in fracture healing[J]. Int Orthop, 2011,35(9) : 1271-1280.
  • 8Kanakaris NK, Petsatodis G, Tagil M, et al. Is there a role for bone morphogenetic proteins in ostoporotie fractures? [J]. Inju- ry,2009,40(Suppl 3) :S21-S26.
  • 9Ghosh-Choudhury N, Mandal CC, Choudhury GG. Statin-induced Ras activation integrates the phosphatidylinositol 3-kinase signal to Akt and MAPK for bone morphogenetic protein-2 expression in osteoblast differentiation[J]. J Biol Chem, 2007, 282 (7): 4983- 4993.
  • 10Chen PY, Sun JS, Tsuang YH, et a l. Simvastatin promotes osteo- blast viability and differentiation via Ras/Smad/Erk/BMP-2 sig- naling pathway[J]. Nutr Res, 2010,30 (3) : 191-199.

引证文献2

二级引证文献12

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部