摘要
Type 2 T helper （Th2） inflammation underlies common clinical disor-ders such as asthma, eczema and other atopic diseases. CD4＋ regulatory T cells （Tregs） suppress over-exuberant immune responses in a variety of inflammatory microenvironments, including Th2 con- ditions. The transcription factor fork- head box protein 3 （Foxp3） controls Treg development, function and main- tenance; however,
Type 2 T helper （Th2） inflammation underlies common clinical disor-ders such as asthma, eczema and other atopic diseases. CD4＋ regulatory T cells （Tregs） suppress over-exuberant immune responses in a variety of inflammatory microenvironments, including Th2 con- ditions. The transcription factor fork- head box protein 3 （Foxp3） controls Treg development, function and main- tenance; however,
