2Kaneto H,Matsuoka TA,Katakami N,et al.Oxidativestress and the JNK pathway are involved in the developmentof type 1 and type 2 diabetes[J].Curr Mol Med,2007,7(7):674-686.
6Milovanovic M.IL-33/ST2 axis in inflammation and immunopa- thology[J].Immunol Res,2012,52(1-2): 89-99.
7Miller AM.Soluble ST2 Associates with Diabetes but Not Estab- lished Cardiovascular Risk Factors: A New Inflammatory Pathway of Relevance to Diabetes?[J].PLoS ONE,2012,7(10):e47830.
8Dallmeier D.Addition of inflammatory biomarkers did not imp- rove diabetes prediction in the community: the fiamingham heart study[J].J Am Heart Assoc,2012,1(4):e000869.
2Borsello T, Forloni G. JNK signalling: a possible target to prevent neurodegeneration. Curr Pharm Des, 2007,13 ( 18 ) : 1875-1886.
3Li Y,Batra S, Sassano A, et al. Activation of mitogen-activated protein kinase kinase (MKK) 3 and MKK6 by type Ⅰ interferons. J Biol Chem,2006,281 (15) : 10651.
4Abell AN, Granger DA, Johnson GL MEKK4 stimulation of p38 and JNK activity is negatively regulated by GSK3beta. J Biol Chem J, 2007,282 ( 42 ) : 30476-30484.
5Bastard JP, Maachi M, Lagathu C, et al. Recent advances in the relationship between obesity, inflammation, and insulin resistance. Eur Cytokine Netw,2006,17 ( 1 ) :4-12.
6Muniyappa H, Das KC . Activation of c-Jun N-terminal kinase (JNK) by widely used pecific p38 MAPK inhibitors SB202190 and SB203580: a MLK-3-MKKT-dependent mechanism. Cell Signal, 2008,20 (4) :675-683.
7Hou N, Torri S, Saito N. Reactive oxygen species-mediated pancreatic beta-cell death is regulated by interactions between stress-activated protein kinases, p58 and c-Jun N-terminal kinase, and mitogen -activated protein kinase phosphatases. Endocrinology, 2008, 149 (4) : 1654-1665.
8He T, Stepulak A, Holmstrom TH, et al. The intermediate filament protein keratin 8 is a novel cytoplasmic substrate for c-Jun N-terminal kinase. J Biol Chem, 2002, 277 ( 13 ) : 10767-10774.
9Johnson GL, Lapadat R. Mitogen-activated protein kinase pathways mediated by ERK, JNK, and p38 protein kinase. Science, 2002, 298:1911-1912.
10Sharfi H, Eldar-Finkelman H. Sequential phosphorylation of insulin receptor substrate-2 by glycogen synthase kinase-3 and c-Jun NH2-terminal kinase plays a role in hepatic insulin signaling. Am J Physiol Endocrinol Metab, 2008, 294 (2) :E307- E315.