期刊文献+

含钛高强带钢中心分层样拉伸过程断口分析 被引量:5

Analysis on Tensile Fracture With Centerline De-Lamination Defect of Ti-Bearing High Strengthen Steel Strip
原文传递
导出
摘要 针对高强焊接结构钢HG70C带钢在力学性能检验中产生拉伸样断口中心分层缺陷,试验设计获得不同拉伸应力的试样,并对比分析未分层试样。通过电镜观察拉伸样断口截面中心偏析带组织和裂纹的演变,从微观上分析了拉伸样断口心部分层微裂纹的渐进演变过程。研究表明,中心处珠光体组织随着变形伸长扩展,与铁素体界面没有产生微孔。分层断口微裂纹是在超过屈服强度后逐步产生的,且在带钢中心厚度位置存在过多连铸偏析的铌钛夹杂物和MnS带边缘处诱发微裂纹。随着拉伸应力逐渐增大,这些微裂纹相互扩展长大形成了贯通平台,最终产生层状分离断口。 Serious centerline de-lamination fractures appeared in hot rolled strip of high-strength structural steel HGTOC during tensile test. Specimens under different stress levels were designed for comparison with normal speci- men. The formation of centerline cracks of the test specimens was studies by optical microscopy and scanning elec- tron microscopy with energy dispersive spectroscopy. Progressive failure for centerline lamination fracture was ana- lyzed~ furthermore the cracking mechanism of de-lamination tensile fracture was described. The results indicated that the pearlite colonies of centerline segregation banding were not the main reason of centerline lamellar fracture. The micro-crack was observed around the inclusions which existed in abundance in the center area. With increasing tensile stress, the micro-cracks coalesced, eventually extended to lamellar cleavage fracture.
出处 《物理测试》 CAS 2014年第2期47-50,共4页 Physics Examination and Testing
关键词 含钛高强钢 分层断口 铌钛夹杂物 Ti-bearing high strengthen steel lamination fracture Nb-Ti inclusion
  • 相关文献

参考文献7

二级参考文献45

  • 1李明,范益.船板拉伸断口分层原因分析[J].南钢科技与管理,2010(1):9-12. 被引量:2
  • 2赵路遇,邢建东,王任甫,徐科,姚上卫.连铸钢板中心偏析及其对组织和韧性的影响[J].钢铁,2005,40(11):62-66. 被引量:17
  • 3韩炯,高亮.高强船板拉伸试验断口分层的原因分析[J].宽厚板,2006,12(1):30-32. 被引量:35
  • 4Zender L, Kuhnel F, Kock R, Manns M, Kubicka S. VP22-mediated intercellular transport of p53 in hepatoma cells in vitro and in vivo. Cancer Gene Ther 2002;9:489-496.
  • 5Post LE. Selectively replicating adenoviruses for cancer therapy: an update on clinical development. Curr Opin Investig Drugs 2002;3:1768-1772.
  • 6Roth JA, Nguyen D, Lawrence DD, Kemp BL, Carrasco CH,Ferson DZ, Hong WK, Komaki R, Lee JJ, Nesbitt JC, Pisters KM, Putnam JB, Schea R, Shin DM, Walsh GL, Dolormente MM, Hart CL Martin FD, Yen N, Xu K, Stephens LC, McDonnell TJ, Mukhopadhyay T, Cai D. Retrovirus-mediated wild-typep 53 gene transfer to tumors of patients with lung cancer. Nat Med 1996;2:985-991.
  • 7Gautam A, Waldrep JC, Kleinerman ES, Xu B, Fung YK, T'Ang A, Densmore CL. Aerosol gene therapy for metastatic lung cancer using PEI-p53 complexes. Methods Mol Med 2003;75:607-618.
  • 8Dolivet G, Merlin JL, Barberi-Heyob M. In vivo growth inhibitory effect of iterative wild-type p53 gene transfer in human head and neck carcinoma xenografts using glucosylated polyethylenimine nonviral vector. Cancer Gene Ther 2002;9:708-714.
  • 9Gautam A, Densmore CL, Melton S, Golunski E, Waldrep JC.Aerosol delivery of PEI-p53 complexes inhibits B16-F10 lung metastases through regulation of angiogenesis. Cancer Gene Ther 2002;9:28-36.
  • 10Seki M, Iwakawa J, Cheng H, Cheng PW. p53 and PTEN/MMAC1/TEP1 gene therapy of human prostate PC-3 carcinoma xenograft, using transferrin-facilitated lipofection gene delivery strategy. Hum Gene Ther 2002;13:761-773.

共引文献107

同被引文献30

引证文献5

二级引证文献14

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部