摘要
铜/锌超氧化物歧化酶(Cu,Zn-SOD)能清除植物体内有害的活性氧(ROS),参与植株遭受逆境胁迫时的应激反应等过程。铜分子伴侣(CCS)可以传递铜离子到Cu,Zn-SOD当中,并将其激活生成有活性的酶分子,依赖CCS协助是Cu,Zn-SOD主要的激活途径。植物在铜缺乏的环境下会诱导启动子结合蛋白(SPL7)和小RNA398(miR398)的表达,miR398通过降解编码Cu,Zn-SOD的mRNA抑制Cu,Zn-SOD的生成,从而调控植物体内铜平衡。本文主要对植物Cu,Zn-SOD激活和调控途径进行综述。
Superoxide dismutases （Cu,Zn-SODs） are important antioxidant enzymes that catalyze the disproportionation of superoxide anion to oxygen and hydrogen peroxide to guard cells against superoxide toxicity. The major pathway for activation of copper/zinc SOD （CSD） requiring the CCS copper chaperone to insert copper and activate SOD1 through oxidation of an intramolecular disulfide. Expression of miR398 and SPL7 （for SQUAMOSA promoter binding protein-like7） are induced in response to copper deficiency and miR398 is involved in the degradation of mRNAs encoding copper/zinc superoxide dismutase. This paper reviewed on plant Cu, Zn-SOD activation and regulatory pathways.
出处
《热带农业科学》
2014年第3期55-61,共7页
Chinese Journal of Tropical Agriculture
基金
国家自然科学基金项目(No.31300497)
华侨大学高层次人才科研启动基金项目
