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注射干扰素α致寻常型银屑病加重2例 被引量：2

The clinical condition of psoriasis got worse after injection of IFN-a in two patients
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摘要 本文报道2例青年男性寻常型银屑病患者,因注射干扰素α后致银屑病加重。 Two young male patients with psoriasis, whose clinical condition got worse after injection of IFN-α, are presented.
作者 秦佩佩 王广进 颜潇潇
机构地区 山东省医学科学院 济南大学山东省医学科学院医学与生命科学学院 山东省皮肤病性病防治研究所
出处 《中国麻风皮肤病杂志》 2014年第5期301-302,共2页 China Journal of Leprosy and Skin Diseases
关键词 银屑病 干扰素Α psoriasis IFN-α
分类号 R758.63 [医药卫生—皮肤病学与性病学]
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参考文献12

  • 1赵辨.中国临床皮肤病学.南京:江苏科学技术出版社,2009.
  • 2Rongioletti F, Fiorucci C, Parodi A. Drug induced/aggravated psoriasis. J Rheumatol Suppl, 2009,83 : 59-61.
  • 3Ketikoglou I, Karatapanis S, Elefsiniotis I, et al. Extensive pso- riasis induced by pegylated interferon alpha- 2b treatment for chronic hepatitis B. Eur J Dermatol, 2005,15 (2) : 107-109.
  • 4Lowes MA, Kikuchi T, Fuentes-Duculan J, et al. Psoriasis vul- garis lesions contain discrete populations of Thl and Th17 T cells. J Invest Dermatol, 2008,128 : 1207-1211.
  • 5Nograles KE, Zaba LC, Guttman-Yassky E, et al. Th17 cyto- kines inter-leukin (IL)- 17 and IL-22 modulate distinct in- ammatory and keratino-cyte-response pathways. Br J Dermatol, 2008,159 : 1092-1102.
  • 6Uyemura K, Yamamura M, Fivenson DF, et al. The cytokine network in lesional and lesion-free psoriatic skin is character- ized by a T-helper type 1 cell-mediated response. J Investig Dermatol, 1993,101:701-705.
  • 7Nestle FO, Conrad C, Tun-Kyi A, et al. Plasmacytoid preden- dritic cells initiate psoriasis through interferon-alpha production. J Exp Med,2005,202: 135-143.
  • 8Afshar M, Martinez AD, Gallo RL, et al. Induction and exacer- bation of psoriasis with Interferonalpha therapy for hepatitis C : A review and analysis of 36 cases.J Eur Acad Dermatol Venereol, 2013,27(6) :771-778.
  • 9Eriksen KW, Lovato P, Skov L, et al. Increased sensitivity to interferon-[ alpha I in psoriatic T cells. J Investig Dermatol, 2005,125:936-944.
  • 10孙会,徐丽敏.干扰素γ与银屑病[J].中国麻风皮肤病杂志,2008,24(3):213-215. 被引量：7

二级参考文献13

  • 1Lew W, Bowcock AM, Krueger JG. Psoriasis vulgaris: cutaneous Lymphoid tissue supports T - cell activation and ‘type 1' inflammatory gene expression. Trends Immunol 2004;25:295- 305.
  • 2Piskin G, Sylva - Steenland RMR, Bos JD, et al. T cells in psoriatic lesional skin that survive conventional therapy with NB- UVB radiation display reduced IFN-γ expression. Arch Dermatol Res 2004;295:509- 516.
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  • 6Chen SH, Arany I, Apisarnthanarax N, et al. Response of keratinocytes from normal and psoriatic epidermis to interferon - gamma differs in the expression of zinc - alpha (2) - glycoprotein and cathepsin D. FASEB J 2000;14:565- 571.
  • 7Grone A. Keratinocytes and cytokines. Vet Immunol Immunopathol 2002;88:1 - 12.
  • 8Kieper WC, Tan JT, Bondi - Boyd B, et al. Overexpression of interleukin (IL) -7 leads to IL- 15 independent generation of memory phenotype CD8^+ T cells. J Exp Med 2002; 195:1533 - 1539.
  • 9Tan JT, Ernst B, Kieper WC, et al. lnterleukin (IL) - 15 and Ⅱ, -7 jointly regulate homeostatic proliferation of memory phenotype CD8^+ cells but are not required for memory phenotype CD4^+ cells. J Exp Med 2002; 195: 1523- 1532.
  • 10Wald O, Weiss ID, Wald H, et al. IFN-γ acts on T cells to induce NK cell mobilization and accumulation in target organs. J Immunol 2006 ; 176: 4716 - 4729.

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中国麻风皮肤病杂志
2014年 第5期

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