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神经营养因子对损伤的SGNs保护的研究进展 被引量:1

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摘要 螺旋神经节细胞(spiral ganglion neuron,SGNs)作为听觉神经传导通路的初级神经元,足够数量正常形态的SGN对听功能的维持是必要的。SGNs属高度分化终末细胞,受损后难以修复,强噪声、耳毒性药物、缺氧等多种因素都有可能造成不同程度的耳蜗毛细胞不可逆的缺失,从而引发SGN的凋亡。
出处 《临床医学》 CAS 2014年第6期119-122,共4页 Clinical Medicine
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