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内毒素与酒精性肝病 被引量:4

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作者 王清图
出处 《中国微循环》 2001年第1期76-77,共2页 Journal of Chinese Microcirculation
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参考文献7

  • 1[1]Thurman R, Bradford BU, Limuro Y, et al. The role of gut-derived bacterial and free radicals in alcohol- induced liver injury [J]. J Gastroenterol Hepatol. 1998,13:S39
  • 2[2]Su GL, Rahemtulla A, Thomas P, et al. CD14 and lipopolysaccharide binding protein expression in a rat model of alcoholic liver disease [J]. Am J Pathol. 1998,152:841
  • 3[3]Adachi Y, Bradford BU, Wenshi G, et al. Inactivation of Kupffer cells prevent early alcohol-induced liver injury [J]. Hepatology. 1994,20:453
  • 4[4]Yin M, Kono H, Bradford BU, et al. Development of a new enteral mouse model using knockout technology to study alcohol- induced liver injury: movement of TNF- alpha {Abstract 635} [J]. Hepatology. 1998,28:321A
  • 5[5]Kroemer G, Dallaporta B, Resche- Rignon M. The mitochondrial death/life regulator in apoptosis and necrosis [J]. Annu Rev Physiol. 1998,60: 619
  • 6[6]arvelainen HA, Fang C, Ingelman- Sundberg M, et al. Effect of chronic coadministration of endotoxin and ethanol on rat liver pathology and proinflammatory and anti- inflammatory cytokines [J]. Hepatology. 1999,29(5):1503
  • 7[7]Enomoto N, Ikejima K, Brandford B, et al. Alcohol causes both tolerance and sensitization of rat Kupffer cells via mechanism dependent on endotoxin[J]. Gastroenterology. 1998,115:443

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