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心房颤动时心房肌钾、钙离子通道重构 被引量:3

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出处 《中华心律失常学杂志》 2001年第3期181-183,共3页 Chinese Journal of Cardiac Arrhythmias
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参考文献9

  • 1van Wagoner DR,Pond AL,Lamorgese M,et al.Atrial L-type Ca2 + currents and human atrial fibrillation[].Circulation Research.1999
  • 2van Wagoner DR. Pond AL,McCathy PM,et al.OutwardK+ current densities and Kv1.5 expression are reduced inchronic human atrial fibrillation[].Circulation Research.1997
  • 3Wang Z,Feng J,Shi H.Potential molecular basis of differentphysiological properties of the transient outward K + current inrabbit and human atrial myocytes[].Circulation Research.1999
  • 4Li GR,Nattle S.Properties of human atrial Ica at physiologicaltemperatures and relevance to action potential[].American Journal of Physiology.1997
  • 5Tieleman RG,Dc Langen CDJ,van Gelder IC. et al.Vera-pamil reduces tachycardia-induced electrical remodeling of theatria[].Circulation.1999
  • 6Ohkusa T,Ueyama T,Yamada J,et al.Alterations in car-diac sarcoplasmic reticulum Ca2+ regulatory proteins in the a-trial tissue of patients with chronic atrial fibrillation[].Journal of the American College of Cardiology.1999
  • 7Yue L,Melnyk P,Gaspo R,et al.Molecular mechanismsunderlying ionic remodeling in a dog of model atrial fibrillation[].Circulation Research.1999
  • 8Fateh S,Benandeau A,Thibault B,et al.The T-type Ca2+channel blocker mibefradil prevents the development of a sub-strate for atrial fibrillation by tachycardia-induced atrial remod-eling in dogs[].Circulation.1999
  • 9Yue L,Feng J,Gaspo H,et al.Ionic remodeling underlyingaction potential changes in a canine model of atrial fibrillation[].Circulation Research.1997

同被引文献26

  • 1Shivkumar K,Weiss JN.Atrial fibrillation:from cells to computers[J]. Cardiovasc Res, 2001, 52(1):171- 173.
  • 2Yue L, Feng J, Gaspo R, et al. Ionic remodeling underlying action potential changes in a canine model of atrial fibrillation [J]. Circ Res, 1997, 81(4): 512-525.
  • 3Van Wagoner DR. Pharmacologic relevance of K + channel remodeling in atrial fibrillation[J]. J Mol Cell Cardiol, 2000,32(9): 1763 - 1766.
  • 4Brandt MC, Priebe L, Bohle T, et al. The ultra- rapid and the transient outward K + current in human atrial fibrillation.Their possible role in postoperative atrial fibrillation[J]. J Mol Cell Cardiol, 2000, 32(10): 1885-1896.
  • 5Van Wagoner DR, Pond AL, McCarthy PM, et al. Outward K + currents and Kv1. 5 K + channels are reduced in chronic human atrial fibrillation[J]. Circ Res, 1997, 80(6): 772 -781.
  • 6Grammer JB, Bosch RF, Kuhlkamp V, et al. Molecular remodeling of Kv 4.3 potassium channels in human atrial fibrillation[J]. J Cardiovasc Electrophysiol, 2000, 11(6): 626-633.
  • 7Lai LP, Su MJ, Lin JL, et al. Down - regulation of L - type calcium channel and sarcoplasmic reticular Ca2 + - ATPase mRNA in human atrial fibrillation without significant change in the mRNA of ryanodine receptor, calsequestrin and phospholamban[J].J Am Coll Cardiol,1999,33(5):1231-1237.
  • 8Bosch RF, Zeng X, Grammner JB, et al. Ionic mechanisms of electrical remodeling in human atrial fibrillation [ J]. Cardiovasc Res, 1999, 44(1): 121-131.
  • 9Workman AJ, Kane KA, Rankin AC. The contribution of ionic currents to changes in refractoriness of human atrial myocytes associated with chronic atrial fibrillation[J]. Cardiovasc Res, 2001, 52(2): 226-235.
  • 10Courtemanche M,Ramirez RJ,Nattel S.Ionic targets for drug therapy and atrial fibrillation-induced electrical remodeling:insights from a mathematical model[J].Cardiovasc Res,1999,42(2):477-489.

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