期刊文献+

脑梗塞后特殊的磁共振成像表现 被引量:2

Research in anal-rectal biofeedback curer
下载PDF
导出
摘要 目的 :分析脑梗塞后MRI短T1 信号的分布、表现特点 ,对其可能的机制进行初步的分析和预测。方法 :回顾性分析105例脑梗塞灶直径>2cm的脑梗塞病人的MRI资料 ,扫描时病程在1d~120d范围内。结果 :105例脑梗塞病人中出现短T1 信号者占48.57 %(51/105)。脑梗塞后短T1 信号最早见于发病后3d内 ,高峰期为发病后7~30d ,短T1 信号的出现与病程呈正相关 (r=0.41 ,P<0.01)。脑梗塞后短T1 信号可发生在皮质区 (59 % )、皮质下区(31 % )及基底节区 (10%) ;表现为线状 (49 % )、斑片状(37%)和斑点状(14%)。结论 :缺血性脑梗塞后短T1 信号的出现与病程呈正相关 ,高峰期为病程第7d~30d ;其产生的病理生理学机制可能为再灌注损伤、皮质层状坏死。 Objective: Analysing the use of biofeedback training in order to treat the encopresis caused by Congenital anal-rectal malformation and anus trauma. Methods: Activating the patients' latent ability by biofeedback training( BFT),which make them defecate independently. Results: The biofeedback training(BFT) not only can in crease the patients' self-confidence, but also can improve their life quality. Conlusion: BFT is an effective curarive means for patients who have suffered from encopresis.
出处 《天津医科大学学报》 2001年第2期219-222,共4页 Journal of Tianjin Medical University
基金 天津医科大学科研基金资助项目(99KY37)
关键词 缺血性脑梗塞 磁共振成像 皮质层状坏死 再灌注损伤 Sensor Peristalsis Rectum Biofeedback
  • 相关文献

参考文献5

  • 1[1]Komiyama M, Nakajima H, Nishikawa M, et al. Serial MR observation of cortical laminar necrosis caused by brain infarction[J ]. Neuroradiology, 1998, 40:771
  • 2[2]Boyko OB, Burger PC, Shelburne JD, et al. Non - heme mechanisms for T1 shortening: pathology, CT, and MR elucidation [J]. Ajnr. 1992, 13:1439
  • 3[3]Valanne L, Paetau A, Suomalainen A, et al. Laminar cortical necrosis in MELAS syndrome: MR and neuropathological observations[J ]. Neuropediatrics, 1996, 27(3): 154
  • 4[4]Fujioka M, Taoka T, Matsuo Y, et al. Delay ischemic hyperintensity on T1 -Weighted MRI in the caudoputamen cerebral cortex of humans after spectacular shrinking deficit[J]. Stroke. 1999, 30:1038
  • 5[5]Fujioka M, Taoka T, Mastsuo Y, et al. Brief ischemic cerebral infarction changes[J]. Stroke. 1999, 30:1042

同被引文献29

  • 1全冠民,袁涛,刘怀军.急性脑梗死出血性转化及MRI评估[J].国外医学(临床放射学分册),2006,29(5):311-314. 被引量:20
  • 2Siskas N,Lefkopoulos A,Ioannidis I,et al.Conical laminar necrosis in brain infarcts:series MRI.Neuroradiology,2003,45(4):283-288.
  • 3Donaire A,Carreno M,Gomez B,et al.NeurolNeurosurg..Psychiatry,2006,77(1):104-106.
  • 4任双燕 张云亭 刘松龄.脑缺血后T1WI产生高信号的病理基础[J].中医药发展与人类健康,2006,8(1):904-907.
  • 5Boyko OB,Burger PC,Shelbume JD,et al.Non-hememechanisms for T1 shortening:pathology,CT and M R elucidation.A jnr,1992,13(1):1439.
  • 6Sawada H,Udaka F,Seriu N,et al.MRI demonstration of cortical laminar necrosis and delayed whitematter injury in anoxic encephalopathy.Neuroradiology,1990,32 (6):319-321.
  • 7Nabatame H,Fujimoto N,et al.High intensity areas on nonconwast T1-weighted MR images in cerebral infarction.J Comput Assist Tomogr,1990,14 (4):521-526.
  • 8David M, Greer MD, Walter J. et al. Magnetic resonance im aging improves detection of intracerebral hemorrhage over computed tomography after intra-arterial thrombolysis [J]. Stroke, 2004, 35: 491-495.
  • 9Latchaw RE, Alberts MJ, Lev MH, et al. Recommendations for imaging of acute ischemic stroke: a scientific statement from the american heart association [J]. Stroke, 2009, 40:3646-3678.
  • 10Reichenbach JR, Haacke EM. High-resolution BOLD venographic imaging: a window into brain function [J]. NMR Biomed, 2001, 14:453-467.

引证文献2

二级引证文献27

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部