1[1]Zhou Y, Richardson JS, Mombourquette MJ, et al. Free radical formationin autops samples of Alzheimer and control cortex. Neurosci Lett, 1995,195: 89-92
2[2]Palmer AM,Burns MA.Selective increse in lipid peroxidation in the infe-rior temporal cortex in Alzheimer' s disease. Brain Res, 1994,645: 338-342
3[3]Subbarao KV, Richardson JS, Ang LC. Autopsy samples of Alzheimer' scortex show increased lipid peroxidation in vitro. J Neuroehem, 1990,55:342-345
4[4]Balazs L, Leon M. Evidence of an oxidation challege in the Alzheimer'sbrain. Neurochem Res, 1994,19:1131-1137
5[5]Andorn AC, Britton RS, Bacon BR. Evidence that lipid peroxidation andtotal iron are increased in Alzheimer's brian. Neurobiol Aging, 1990,11:316
7[7]Gutteridge JM, Quinlan GJ, Clark I, et al. Aluminium salts accelerate per-oxidation of membrane lipids stimulated by iron salts. Biochim BiophysActa, 1985,835: 441-447
8[8]Gotz ME, Fischer P, Gsell W, et al. Platelet monoamine oxidse B activityin dementia: a 4 year follow up. Dementia, 1998,9 :74-77
9[9]Christen Y. Oxidative stress and Alzheimer Disease. Am J Clin Nutr,2000,71(2) :621S-629S
10[10]Loyel MA, Ehmann WD, Butler SM, et al. Elevated thiobarbituic acid-re-active substance and antioxidant enzyme activity in the brain of Alzheimer's disease. Neurology, 1995,45:1594-1601
4Wenk J, Brenneisen P, Wlaschek M, et al.Stable overexpression of manganese superoxide dismutase in mitochondria identifies hydrogen peroxide as a major oxidant in the AP-l-mediated induction of matrix-degrading metalloprotease-1 [J]. Biol Chem, 1999,274(36) :25869-25876.
5Shigenaga M K,Hagen T M,Ames B N,et al. Oxidative damage and mitochondrial decay in aging[J]. Proc Natl Acad Sci USA,1994,91(23):10771-10778.
6Babcock G T, Wikstrom M. Oxygen activation and the conservation of energy in cell respiration [J]. Nature, 1992,356 (6367):301-309.
7Sobll S. Thyroid hormone action on mitochondrial energy transfer [J]. Biochim Biophys Acta,1993, 1144(1) :1-16.
8Cassarino D S, Parks J K, Parker W D, et al. The parkinsonian neurotoxin MPP^+ opens the mitochondrial permeability transition pore and releases cytochrome C in isolated mitochondria via an oxidative mechanism[J]. Biochim Biophys Acta, 1999, 1453(1):49-62.
9Lass A, Sohal B H, Weindruch R, et al.Caloric restriction prevents age-associated accrul of oxidative damage to mouse skeletal muscle mitochondrial[J].Free Rad Biol Med, 1998, 25:1089-1097.
10Kristal B, Park B, Yu B P. Antioxidants reduce peroxyl-mediated inhibition of mitochondrial [J]. Free Rad Biol Med, 1994,216 : 653-660.